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本文引用的文献

1
Loss of halophytism by interference with SOS1 expression.通过干扰SOS1表达导致盐生植物特性丧失。
Plant Physiol. 2009 Sep;151(1):210-22. doi: 10.1104/pp.109.137802. Epub 2009 Jul 1.
2
Learning from evolution: Thellungiella generates new knowledge on essential and critical components of abiotic stress tolerance in plants.从进化中学习:盐芥为植物非生物胁迫耐受性的必需和关键成分带来新知识。
Mol Plant. 2009 Jan;2(1):3-12. doi: 10.1093/mp/ssn094.
3
Phosphorylation of SOS3-LIKE CALCIUM BINDING PROTEIN8 by SOS2 protein kinase stabilizes their protein complex and regulates salt tolerance in Arabidopsis.SOS2蛋白激酶对类SOS3钙结合蛋白8的磷酸化作用可稳定其蛋白复合物,并调控拟南芥的耐盐性。
Plant Cell. 2009 May;21(5):1607-19. doi: 10.1105/tpc.109.066217. Epub 2009 May 15.
4
Emerging roles of alkali cation/proton exchangers in organellar homeostasis.碱金属阳离子/质子交换体在细胞器稳态中的新作用。
Curr Opin Cell Biol. 2007 Aug;19(4):483-92. doi: 10.1016/j.ceb.2007.06.001. Epub 2007 Jul 23.
5
Cyclic nucleotide-gated channels in plants.植物中的环核苷酸门控通道。
FEBS Lett. 2007 May 25;581(12):2237-46. doi: 10.1016/j.febslet.2007.02.017. Epub 2007 Feb 16.
6
AtNHX8, a member of the monovalent cation: proton antiporter-1 family in Arabidopsis thaliana, encodes a putative Li/H antiporter.AtNHX8是拟南芥单价阳离子:质子反向转运蛋白-1家族的成员,编码一种假定的锂/氢反向转运蛋白。
Plant J. 2007 Feb;49(4):718-28. doi: 10.1111/j.1365-313X.2006.02990.x. Epub 2007 Jan 18.
7
Conservation of the salt overly sensitive pathway in rice.水稻中盐超敏感途径的保守性。
Plant Physiol. 2007 Feb;143(2):1001-12. doi: 10.1104/pp.106.092635. Epub 2006 Dec 1.
8
Suppression of Arabidopsis vesicle-SNARE expression inhibited fusion of H2O2-containing vesicles with tonoplast and increased salt tolerance.拟南芥囊泡-SNARE 表达的抑制会抑制含过氧化氢的囊泡与液泡膜的融合,并提高耐盐性。
Proc Natl Acad Sci U S A. 2006 Nov 21;103(47):18008-13. doi: 10.1073/pnas.0604421103. Epub 2006 Nov 13.
9
Transcriptional profiling implicates novel interactions between abiotic stress and hormonal responses in Thellungiella, a close relative of Arabidopsis.转录谱分析揭示了拟南芥的近缘种盐芥中非生物胁迫与激素反应之间的新相互作用。
Plant Physiol. 2006 Apr;140(4):1437-50. doi: 10.1104/pp.105.070508. Epub 2006 Feb 24.
10
Salt stress signaling and mechanisms of plant salt tolerance.盐胁迫信号传导与植物耐盐机制。
Genet Eng (N Y). 2006;27:141-77. doi: 10.1007/0-387-25856-6_9.

SOS1 与盐生植物。

SOS1 and halophytism.

机构信息

Departments of Plant Biology and of Crop Sciences, University of Illinois at Urbana Champaign, Urbana, IL, USA.

出版信息

Plant Signal Behav. 2009 Nov;4(11):1081-3. doi: 10.4161/psb.4.11.9786. Epub 2009 Nov 11.

DOI:10.4161/psb.4.11.9786
PMID:19838069
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2819520/
Abstract

Much is already known about the function and functioning of the three genes that make up the SOS (Salt-Overly-Sensitive) pathway in plants, but recent studies indicate that the linkage between external increases in salinity and stress protection provided by genes SOS1, SOS2 and SOS3 is more complex than previously appreciated. It has recently been shown that the engineered reduced expression of the sodium/proton antiporter SOS1 affected several pathways indicating a role for SOS1 that exceeds its known function as an antiporter. Interference with expression of SOS1, characterized as a sodium/proton antiporter in the halophyte Thellungiella salsuginea converted Thellungiella into an essentially glycophytic species.

摘要

关于构成植物 SOS(盐过度敏感)途径的三个基因的功能和作用,人们已经了解很多,但最近的研究表明,外部盐度增加与 SOS1、SOS2 和 SOS3 基因提供的应激保护之间的联系比以前认为的更为复杂。最近表明,工程改造降低 SOS1 钠/质子反向转运蛋白的表达影响了几个途径,表明 SOS1 的作用超出了其作为反向转运蛋白的已知功能。在盐生植物 Thellungiella salsuginea 中,干扰 SOS1 的表达,其特征为一种钠/质子反向转运蛋白,将 Thellungiella 转化为基本上的嗜盐植物。