Department of Physiology, School of Medicine-Instituto de Investigación Sanitaria (IDIS), University of Santiago de Compostela, 15782 Santiago de Compostela, Spain.
Obes Rev. 2010 Mar;11(3):185-201. doi: 10.1111/j.1467-789X.2009.00669.x. Epub 2009 Oct 21.
The central nervous system (CNS) is crucial in the regulation of energy homeostasis. Many neuroanatomical studies have shown that the white adipose tissue (WAT) is innervated by the sympathetic nervous system, which plays a critical role in adipocyte lipid metabolism. Therefore, there are currently numerous reports indicating that signals from the CNS control the amount of fat by modulating the storage or oxidation of fatty acids. Importantly, some CNS pathways regulate adipocyte metabolism independently of food intake, suggesting that some signals possess alternative mechanisms to regulate energy homeostasis. In this review, we mainly focus on how neuronal circuits within the hypothalamus, such as leptin- ghrelin-and resistin-responsive neurons, as well as melanocortins, neuropeptide Y, and the cannabinoid system exert their actions on lipid metabolism in peripheral tissues such as WAT, liver or muscle. Dissecting the complicated interactions between peripheral signals and neuronal circuits regulating lipid metabolism might open new avenues for the development of new therapies preventing and treating obesity and its associated cardiometabolic sequelae.
中枢神经系统(CNS)在能量稳态调节中起着至关重要的作用。许多神经解剖学研究表明,白色脂肪组织(WAT)受交感神经系统支配,交感神经系统在脂肪细胞脂质代谢中起着关键作用。因此,目前有大量报道表明,CNS 信号通过调节脂肪酸的储存或氧化来控制脂肪量。重要的是,一些 CNS 途径在不依赖于食物摄入的情况下调节脂肪细胞代谢,这表明一些信号具有调节能量稳态的替代机制。在这篇综述中,我们主要关注下丘脑内的神经元回路如何,如瘦素-ghrelin-和抵抗素反应神经元,以及黑皮质素、神经肽 Y 和大麻素系统,在外周组织(如 WAT、肝脏或肌肉)中对脂质代谢发挥作用。解析调节脂质代谢的外周信号和神经元回路之间复杂的相互作用,可能为预防和治疗肥胖及其相关心血管代谢后果的新疗法的发展开辟新途径。
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