Opiates, sleep, and pain: the adenosinergic link.
作者信息
Moore Jason T, Kelz Max B
出版信息
Anesthesiology. 2009 Dec;111(6):1175-6. doi: 10.1097/ALN.0b013e3181bdfa2e.
Abstract
摘要
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本文引用的文献
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Opioid-induced decreases in rat brain adenosine levels are reversed by inhibiting adenosine deaminase.通过抑制腺苷脱氨酶可逆转阿片类药物引起的大鼠脑内腺苷水平降低。
Anesthesiology. 2009 Dec;111(6):1327-33. doi: 10.1097/ALN.0b013e3181bdf894.
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Opioidergic projections to sleep-active neurons in the ventrolateral preoptic nucleus.向腹外侧视前核中睡眠活跃神经元的阿片能投射。
Brain Res. 2008 Dec 15;1245:96-107. doi: 10.1016/j.brainres.2008.09.043. Epub 2008 Sep 24.
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Inhibition of nociceptors by TRPV1-mediated entry of impermeant sodium channel blockers.通过TRPV1介导的非渗透性钠通道阻滞剂的进入来抑制伤害感受器。
Nature. 2007 Oct 4;449(7162):607-10. doi: 10.1038/nature06191.
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An SCN9A channelopathy causes congenital inability to experience pain.一种SCN9A通道病导致先天性无痛觉。
Nature. 2006 Dec 14;444(7121):894-8. doi: 10.1038/nature05413.
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Sleep and GABA levels in the oral part of rat pontine reticular formation are decreased by local and systemic administration of morphine.局部和全身给予吗啡会降低大鼠脑桥网状结构口腔部的睡眠和γ-氨基丁酸水平。
Neuroscience. 2007 Jan 5;144(1):375-86. doi: 10.1016/j.neuroscience.2006.09.007. Epub 2006 Oct 19.
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Morphine inhibits acetylcholine release in rat prefrontal cortex when delivered systemically or by microdialysis to basal forebrain.当通过全身给药或微透析法将吗啡输送至基底前脑时，它会抑制大鼠前额叶皮质中乙酰胆碱的释放。
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5-HT4(a) receptors avert opioid-induced breathing depression without loss of analgesia.5-HT4(a)受体可避免阿片类药物引起的呼吸抑制，且不丧失镇痛作用。
Science. 2003 Jul 11;301(5630):226-9. doi: 10.1126/science.1084674.
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The sleep switch: hypothalamic control of sleep and wakefulness.睡眠开关：下丘脑对睡眠和觉醒的控制
Trends Neurosci. 2001 Dec;24(12):726-31. doi: 10.1016/s0166-2236(00)02002-6.
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Adenosine: a mediator of the sleep-inducing effects of prolonged wakefulness.腺苷：长时间清醒诱导睡眠效应的一种介质。
Science. 1997 May 23;276(5316):1265-8. doi: 10.1126/science.276.5316.1265.
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Chronic morphine administration in cats: effects on sleep and EEG.猫长期给予吗啡：对睡眠和脑电图的影响。
Pharmacol Biochem Behav. 1989 Feb;32(2):519-26. doi: 10.1016/0091-3057(89)90191-3.
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