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衰老的氧化应激理论更新:氧化应激在衰老或健康衰老中起作用吗?

Update on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?

机构信息

The Sam and Ann Barshop Institute for Longevity and Aging Studies, The University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA.

出版信息

Free Radic Biol Med. 2010 Mar 1;48(5):642-55. doi: 10.1016/j.freeradbiomed.2009.12.015. Epub 2009 Dec 28.

Abstract

The oxidative stress theory of aging predicts that manipulations that alter oxidative stress/damage will alter aging. The gold standard for determining whether aging is altered is life span, i.e., does altering oxidative stress/damage change life span? Mice with genetic manipulations in their antioxidant defense system designed to directly address this prediction have, with few exceptions, shown no change in life span. However, when these transgenic/knockout mice are tested using models that develop various types of age-related pathology, they show alterations in progression and/or severity of pathology as predicted by the oxidative stress theory: increased oxidative stress accelerates pathology and reduced oxidative stress retards pathology. These contradictory observations might mean that (a) oxidative stress plays a very limited, if any, role in aging but a major role in health span and/or (b) the role that oxidative stress plays in aging depends on environment. In environments with minimal stress, as expected under optimal husbandry, oxidative damage plays little role in aging. However, under chronic stress, including pathological phenotypes that diminish optimal health, oxidative stress/damage plays a major role in aging. Under these conditions, enhanced antioxidant defenses exert an "antiaging" action, leading to changes in life span, age-related pathology, and physiological function as predicted by the oxidative stress theory of aging.

摘要

衰老的氧化应激理论预测,改变氧化应激/损伤的操作将改变衰老。确定衰老是否改变的金标准是寿命,即改变氧化应激/损伤是否改变寿命?具有旨在直接解决这一预测的抗氧化防御系统遗传操作的小鼠,除了少数例外,寿命没有变化。然而,当这些转基因/敲除小鼠使用会发展出各种与年龄相关的病理的模型进行测试时,它们表现出与氧化应激理论预测的病理进展和/或严重程度的改变:增加氧化应激会加速病理,而减少氧化应激会延缓病理。这些相互矛盾的观察结果可能意味着:(a)氧化应激在衰老中仅起非常有限的作用,如果有的话,但在健康跨度中起主要作用和/或(b)氧化应激在衰老中所起的作用取决于环境。在应激最小的环境中,如在最佳饲养条件下预期的那样,氧化损伤在衰老中几乎不起作用。然而,在慢性应激下,包括降低最佳健康的病理表型,氧化应激/损伤在衰老中起主要作用。在这些条件下,增强的抗氧化防御会产生“抗衰老”作用,导致寿命、与年龄相关的病理和生理功能的改变,正如衰老的氧化应激理论所预测的那样。

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