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Peroxisome proliferator-activated receptor gamma dances with different partners in macrophage and adipocytes.

作者信息

Scotti Elena, Tontonoz Peter

机构信息

Howard Hughes Medical Institute and Department of Pathology and Laboratory Medicine, University of California, Los Angeles, Los Angeles, CA 99095-1662, USA.

出版信息

Mol Cell Biol. 2010 May;30(9):2076-7. doi: 10.1128/MCB.00171-10. Epub 2010 Feb 22.

DOI:10.1128/MCB.00171-10
PMID:20176804
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2863579/
Abstract
摘要

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本文引用的文献

1
Cell-specific determinants of peroxisome proliferator-activated receptor gamma function in adipocytes and macrophages.脂肪细胞和巨噬细胞中过氧化物酶体增殖物激活受体 γ 功能的细胞特异性决定因素。
Mol Cell Biol. 2010 May;30(9):2078-89. doi: 10.1128/MCB.01651-09. Epub 2010 Feb 22.
2
Genome-wide profiling of PPARgamma:RXR and RNA polymerase II occupancy reveals temporal activation of distinct metabolic pathways and changes in RXR dimer composition during adipogenesis.全基因组范围内PPARγ:RXR和RNA聚合酶II占据情况分析揭示脂肪生成过程中不同代谢途径的时间性激活以及RXR二聚体组成的变化。
Genes Dev. 2008 Nov 1;22(21):2953-67. doi: 10.1101/gad.501108.
3
PPARgamma and C/EBP factors orchestrate adipocyte biology via adjacent binding on a genome-wide scale.过氧化物酶体增殖物激活受体γ(PPARγ)和CCAAT增强子结合蛋白(C/EBP)因子通过全基因组范围内的相邻结合来调控脂肪细胞生物学。
Genes Dev. 2008 Nov 1;22(21):2941-52. doi: 10.1101/gad.1709008.
4
Fat and beyond: the diverse biology of PPARgamma.脂肪及其他:过氧化物酶体增殖物激活受体γ的多样生物学特性
Annu Rev Biochem. 2008;77:289-312. doi: 10.1146/annurev.biochem.77.061307.091829.
5
A histone lysine methyltransferase activated by non-canonical Wnt signalling suppresses PPAR-gamma transactivation.一种由非经典Wnt信号激活的组蛋白赖氨酸甲基转移酶可抑制PPAR-γ的反式激活。
Nat Cell Biol. 2007 Nov;9(11):1273-85. doi: 10.1038/ncb1647. Epub 2007 Oct 21.
6
Macrophage-specific PPARgamma controls alternative activation and improves insulin resistance.巨噬细胞特异性过氧化物酶体增殖物激活受体γ控制替代性激活并改善胰岛素抵抗。
Nature. 2007 Jun 28;447(7148):1116-20. doi: 10.1038/nature05894. Epub 2007 May 21.
7
Combinatorial roles of nuclear receptors in inflammation and immunity.核受体在炎症和免疫中的组合作用。
Nat Rev Immunol. 2006 Jan;6(1):44-55. doi: 10.1038/nri1748.
8
A SUMOylation-dependent pathway mediates transrepression of inflammatory response genes by PPAR-gamma.一条依赖于小泛素样修饰(SUMOylation)的信号通路介导过氧化物酶体增殖物激活受体γ(PPAR-γ)对炎症反应基因的反式抑制作用。
Nature. 2005 Sep 29;437(7059):759-63. doi: 10.1038/nature03988. Epub 2005 Aug 28.
9
Nuclear receptors in macrophage biology: at the crossroads of lipid metabolism and inflammation.巨噬细胞生物学中的核受体:处于脂质代谢与炎症的交叉点
Annu Rev Cell Dev Biol. 2004;20:455-80. doi: 10.1146/annurev.cellbio.20.012103.134432.
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PPAR-gamma dependent and independent effects on macrophage-gene expression in lipid metabolism and inflammation.过氧化物酶体增殖物激活受体γ(PPAR-γ)对脂质代谢和炎症中巨噬细胞基因表达的依赖性和非依赖性作用。
Nat Med. 2001 Jan;7(1):48-52. doi: 10.1038/83336.