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Acta Vet Scand. 1990;31(3):257-65. doi: 10.1186/BF03547538.
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本文引用的文献

1
White liver disease in lambs.羔羊白肝病
Aust Vet J. 1981 Dec;57(12):565-8. doi: 10.1111/j.1751-0813.1981.tb00437.x.
2
White liver disease of sheep.绵羊白肝病
Aust Vet J. 1982 May;58(5):181-4. doi: 10.1111/j.1751-0813.1982.tb00648.x.
3
Hepatic damage in sheep fed annual ryegrass, Lolium rigidum, parasitised by Anguina agrostis and Corynebacterium rathayi.被禾本科粒线虫和拉氏棒状杆菌寄生的、以一年生黑麦草(多花黑麦草)为食的绵羊的肝脏损伤
Res Vet Sci. 1982 Mar;32(2):148-56.
4
Ovine white liver disease.绵羊白肝病
Aust Vet J. 1983 Jul;60(7):219-20. doi: 10.1111/j.1751-0813.1983.tb09590.x.
5
Liver lesions resembling ovine white liver disease in cobalt deficient lambs.钴缺乏的羔羊中出现类似绵羊白肝病的肝脏病变。
Vet Rec. 1984 Sep 29;115(13):325. doi: 10.1136/vr.115.13.325.
6
The pathology of lupinosis of sheep. Gross- and histo-pathology.绵羊羽扇豆中毒的病理学。大体病理学和组织病理学。
Pathol Vet. 1965;2(5):417-45. doi: 10.1177/030098586500200501.
7
Cobalt deficiency in ruminants.
Vet Rec. 1971 Jul 3;89(1):5-12. doi: 10.1136/vr.89.1.5.
8
Chronic poisoning in a lamb grazing Phalaris arundinacea.一只食用虉草的羔羊发生慢性中毒。
Acta Vet Scand. 1985;26(2):286-8. doi: 10.1186/BF03546558.
9
Pyrrolizidine alkaloid poisoning of sheep in New South Wales.
Aust Vet J. 1987 Jun;64(6):164-7. doi: 10.1111/j.1751-0813.1987.tb09674.x.
10
Ovine white-liver disease (OWLD) in Norway: clinical symptoms and preventive measures.挪威的绵羊白肝病:临床症状及预防措施
Acta Vet Scand. 1990;31(1):53-62. doi: 10.1186/BF03547577.

绵羊白肝病(OWLD)。牧草的植物学和化学成分。

Ovine white-liver disease (OWLD). Botanical and chemical composition of pasture grass.

作者信息

Ulvund M J, Pestalozzi M

机构信息

State Veterinary Research Station for Small Ruminants, Sandnes, Norway.

出版信息

Acta Vet Scand. 1990;31(3):257-65. doi: 10.1186/BF03547538.

DOI:10.1186/BF03547538
PMID:2080770
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8133152/
Abstract

The most important grass species on the ovine white-liver disease (OWLD) pastures (S) were Poa spp., Agropyron repens and Lolium perenne, while the control pastures (H), where lambs grew well, consisted of Poa spp., Festuca rubra and Agrostis tenuis. The soil was more acidic on the H pastures as compared with the S pastures. OWLD grass (S grass) contained marginal to deficient amounts of cobalt during the first 2 months of grazing. During 2 years out of 3, the average Co content was slightly lower in the S grass as compared with the content in the H grass. The lowest average grass Co was, however, found during one year in the H grass, in spite of the fact that the H lambs also this year grew well, and were 13 kg heavier than the S lambs after 3 1/2 months on pasture. Results thus indicate that the H lambs some years were subclinically Co deficient, without developing clinical symptoms or OWLD, and that factors other than marginal/deficient grass Co are of importance as to whether OWLD will develop or not. S grass differed from H grass by having significantly lower copper, molybdenum, manganese and zinc content, lower protein N/amid N ratios and higher aluminium and iron contents. According to the results, marginal to deficient grass Co is essential for development of OWLD, but cofactors play a part.

摘要

患有绵羊白肝病(OWLD)的牧场(S)中最重要的草种是早熟禾属、匍匐翦股颖和多年生黑麦草,而羔羊生长良好的对照牧场(H)则由早熟禾属、紫羊茅和细弱翦股颖组成。与S牧场相比,H牧场的土壤酸性更强。在放牧的前两个月,OWLD草(S草)中的钴含量处于边缘水平或不足。在三年中的两年里,S草中的平均钴含量略低于H草中的含量。然而,尽管今年H牧场的羔羊生长良好,且在放牧3个半月后比S牧场的羔羊重13千克,但在某一年的H草中发现了最低的平均草钴含量。因此,结果表明,某些年份的H羔羊存在亚临床钴缺乏,但未出现临床症状或OWLD,并且除了边缘/缺乏的草钴之外,其他因素对于OWLD是否会发生也很重要。S草与H草的不同之处在于,其铜、钼、锰和锌含量显著较低,蛋白质氮/酰胺氮比率较低,铝和铁含量较高。根据结果,边缘或缺乏的草钴对于OWLD的发展至关重要,但辅助因子也起作用。