急性关节炎症期间白细胞介素-1β的激活：NLRP3炎性小体在体内的作用有限。 - Suppr

Interleukin-1β activation during acute joint inflammation: a limited role for the NLRP3 inflammasome in vivo.

作者信息

Joosten Leo A B, Ea Hang-Korng, Netea Mihai G, Busso Nathalie

出版信息

Joint Bone Spine. 2011 Mar;78(2):107-10. doi: 10.1016/j.jbspin.2010.11.004. Epub 2010 Dec 22.

DOI:10.1016/j.jbspin.2010.11.004

PMID:21183380

Abstract

摘要

相似文献

Interleukin-1β activation during acute joint inflammation: a limited role for the NLRP3 inflammasome in vivo.

Joint Bone Spine. 2011 Mar;78(2):107-10. doi: 10.1016/j.jbspin.2010.11.004. Epub 2010 Dec 22.

["The inflammasomes"].

Nihon Rinsho Meneki Gakkai Kaishi. 2011;34(5):346-54. doi: 10.2177/jsci.34.346.

Critical roles of ASC inflammasomes in caspase-1 activation and host innate resistance to Streptococcus pneumoniae infection.

J Immunol. 2011 Nov 1;187(9):4890-9. doi: 10.4049/jimmunol.1100381. Epub 2011 Sep 28.

Critical role for the AIM2 inflammasome during acute CNS bacterial infection.

J Neurochem. 2014 May;129(4):704-11. doi: 10.1111/jnc.12669. Epub 2014 Feb 19.

Interferon-β therapy against EAE is effective only when development of the disease depends on the NLRP3 inflammasome.

Sci Signal. 2012 May 22;5(225):ra38. doi: 10.1126/scisignal.2002767.

The inflammasome sensor, NLRP3, regulates CNS inflammation and demyelination via caspase-1 and interleukin-18.

J Neurosci. 2010 Nov 24;30(47):15811-20. doi: 10.1523/JNEUROSCI.4088-10.2010.

Stress-induced cartilage degradation does not depend on the NLRP3 inflammasome in human osteoarthritis and mouse models.

Arthritis Rheum. 2012 Dec;64(12):3972-81. doi: 10.1002/art.34678.

HMGB1 promotes the activation of NLRP3 and caspase-8 inflammasomes via NF-κB pathway in acute glaucoma.

J Neuroinflammation. 2015 Jul 30;12:137. doi: 10.1186/s12974-015-0360-2.

The purinergic 2X7 receptor participates in renal inflammation and injury induced by high-fat diet: possible role of NLRP3 inflammasome activation.

J Pathol. 2013 Nov;231(3):342-53. doi: 10.1002/path.4237. Epub 2013 Sep 3.

NLRP3 (NALP3, Cryopyrin) facilitates in vivo caspase-1 activation, necrosis, and HMGB1 release via inflammasome-dependent and -independent pathways.

J Immunol. 2009 Aug 1;183(3):2008-15. doi: 10.4049/jimmunol.0900138. Epub 2009 Jul 8.

引用本文的文献

Disulfiram with Cu alleviates dextran sulfate sodium-induced ulcerative colitis in mice.

Theranostics. 2023 May 8;13(9):2879-2895. doi: 10.7150/thno.81571. eCollection 2023.

Where Epigenetics Meets Food Intake: Their Interaction in the Development/Severity of Gout and Therapeutic Perspectives.

Front Immunol. 2021 Sep 17;12:752359. doi: 10.3389/fimmu.2021.752359. eCollection 2021.

A mouse model of MSU-induced acute inflammation suggests imiquimod-dependent targeting of as relevant therapy for gout patients.

Theranostics. 2020 Jan 12;10(5):2158-2171. doi: 10.7150/thno.40650. eCollection 2020.

The Pyroptotic Cell Death Effector Gasdermin D Is Activated by Gout-Associated Uric Acid Crystals but Is Dispensable for Cell Death and IL-1β Release.

J Immunol. 2019 Aug 1;203(3):736-748. doi: 10.4049/jimmunol.1900228. Epub 2019 Jun 17.

Macrophage Recognition of Crystals and Nanoparticles.

Front Immunol. 2018 Jan 29;9:103. doi: 10.3389/fimmu.2018.00103. eCollection 2018.

Hydrogen sulfide inhibits NLRP3 inflammasome activation and reduces cytokine production both and in a mouse model of inflammation.

J Biol Chem. 2018 Feb 16;293(7):2546-2557. doi: 10.1074/jbc.M117.806869. Epub 2017 Dec 26.

Physiological functions and pathogenic potential of uric acid: A review.

J Adv Res. 2017 Sep;8(5):487-493. doi: 10.1016/j.jare.2017.03.003. Epub 2017 Mar 14.

Neutrophil microvesicles resolve gout by inhibiting C5a-mediated priming of the inflammasome.

Ann Rheum Dis. 2016 Jun;75(6):1236-45. doi: 10.1136/annrheumdis-2015-207338. Epub 2015 Aug 5.

AMP-activated protein kinase suppresses urate crystal-induced inflammation and transduces colchicine effects in macrophages.

Ann Rheum Dis. 2016 Jan;75(1):286-94. doi: 10.1136/annrheumdis-2014-206074. Epub 2014 Oct 31.

NLRP3 inflammasome activation and interleukin-1β release in macrophages require calcium but are independent of calcium-activated NADPH oxidases.

Inflamm Res. 2014 Oct;63(10):821-30. doi: 10.1007/s00011-014-0756-y. Epub 2014 Jul 22.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验

Interleukin-1β activation during acute joint inflammation: a limited role for the NLRP3 inflammasome in vivo.

作者信息

出版信息

相似文献

引用本文的文献

文献AI研究员

用中文搜PubMed

文档翻译

Suppr 超能文献

相似文献

引用本文的文献