Multiple death pathways in TNF-treated fibroblasts: RIP3- and RIP1-dependent and independent routes.
作者信息
Zhang Duan-Wu, Zheng Min, Zhao Jing, Li Yuan-Yue, Huang Zhe, Li Zhu, Han Jiahuai
出版信息
Cell Res. 2011 Feb;21(2):368-71. doi: 10.1038/cr.2011.3. Epub 2011 Jan 4.
Abstract
摘要
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本文引用的文献
1
Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation.磷酸化驱动的RIP1-RIP3复合物组装调节程序性坏死和病毒诱导的炎症。
Cell. 2009 Jun 12;137(6):1112-23. doi: 10.1016/j.cell.2009.05.037.
2
Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha.受体相互作用蛋白激酶-3决定细胞对肿瘤坏死因子-α的坏死反应。
Cell. 2009 Jun 12;137(6):1100-11. doi: 10.1016/j.cell.2009.05.021.
3
RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis.RIP3，一种能量代谢调节因子，可将肿瘤坏死因子诱导的细胞死亡从凋亡转变为坏死。
Science. 2009 Jul 17;325(5938):332-6. doi: 10.1126/science.1172308. Epub 2009 Jun 4.
4
Identification of a molecular signaling network that regulates a cellular necrotic cell death pathway.调控细胞坏死性细胞死亡途径的分子信号网络的鉴定。
Cell. 2008 Dec 26;135(7):1311-23. doi: 10.1016/j.cell.2008.10.044.
5
RIP1, a kinase on the crossroads of a cell's decision to live or die.RIP1，一种处于细胞生死抉择十字路口的激酶。
Cell Death Differ. 2007 Mar;14(3):400-10. doi: 10.1038/sj.cdd.4402085.
6
Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis.抑制受体相互作用蛋白介导的坏死中ADP/ATP的交换
Mol Cell Biol. 2006 Mar;26(6):2215-25. doi: 10.1128/MCB.26.6.2215-2225.2006.
7
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury.具有治疗缺血性脑损伤潜力的非凋亡性细胞死亡化学抑制剂。
Nat Chem Biol. 2005 Jul;1(2):112-9. doi: 10.1038/nchembio711. Epub 2005 May 29.
8
JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species.JNK通过增加细胞毒性活性氧的产生来增强肿瘤坏死因子刺激的坏死。
Genes Dev. 2004 Dec 1;18(23):2905-15. doi: 10.1101/gad.1223004. Epub 2004 Nov 15.
9
Regulation of an ATG7-beclin 1 program of autophagic cell death by caspase-8.半胱天冬酶-8对自噬性细胞死亡的ATG7-贝林1程序的调控
Science. 2004 Jun 4;304(5676):1500-2. doi: 10.1126/science.1096645. Epub 2004 May 6.
10
Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation.肿瘤坏死因子诱导的非凋亡性细胞死亡需要受体相互作用蛋白介导的细胞活性氧积累。
J Biol Chem. 2004 Mar 12;279(11):10822-8. doi: 10.1074/jbc.M313141200. Epub 2003 Dec 29.
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