Prion diseases: Infectivity versus toxicity.
作者信息
Wickner Reed B
出版信息
Nature. 2011 Feb 24;470(7335):470-1. doi: 10.1038/470470a.
Abstract
摘要
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本文引用的文献
1
Prion propagation and toxicity in vivo occur in two distinct mechanistic phases.朊病毒在体内的传播和毒性存在于两个不同的机制阶段。
Nature. 2011 Feb 24;470(7335):540-2. doi: 10.1038/nature09768.
2
Getting a grip on prions: oligomers, amyloids, and pathological membrane interactions.掌控朊病毒：寡聚体、淀粉样蛋白及病理性膜相互作用
Annu Rev Biochem. 2009;78:177-204. doi: 10.1146/annurev.biochem.78.082907.145410.
3
Anchorless prion protein results in infectious amyloid disease without clinical scrapie.无锚定朊病毒蛋白导致传染性淀粉样疾病，无临床瘙痒病症状。
Science. 2005 Jun 3;308(5727):1435-9. doi: 10.1126/science.1110837.
4
Cross-linking cellular prion protein triggers neuronal apoptosis in vivo.细胞朊蛋白交联在体内触发神经元凋亡。
Science. 2004 Mar 5;303(5663):1514-6. doi: 10.1126/science.1094273. Epub 2004 Jan 29.
5
A quantitative, highly sensitive cell-based infectivity assay for mouse scrapie prions.一种用于小鼠瘙痒病朊病毒的定量、高灵敏度的基于细胞的感染性检测方法。
Proc Natl Acad Sci U S A. 2003 Sep 30;100(20):11666-71. doi: 10.1073/pnas.1834432100. Epub 2003 Sep 22.
6
Normal host prion protein necessary for scrapie-induced neurotoxicity.正常宿主朊病毒蛋白是羊瘙痒病诱导神经毒性所必需的。
Nature. 1996 Jan 25;379(6563):339-43. doi: 10.1038/379339a0.
7
The scrapie-associated form of PrP is made from a cell surface precursor that is both protease- and phospholipase-sensitive.瘙痒病相关形式的朊蛋白由一种对蛋白酶和磷脂酶敏感的细胞表面前体产生。
J Biol Chem. 1991 Sep 25;266(27):18217-23.
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