Exposure to low level environmental agents: the induction of hormesis.
作者信息
Azzam Edouard I
机构信息
Department of Radiology, UMDNJ - New Jersey Medical School Cancer Center, Newark, NJ, USA.
出版信息
Mutat Res. 2011 Dec 24;726(2):89-90. doi: 10.1016/j.mrgentox.2011.07.008. Epub 2011 Jul 23.
Abstract
摘要
相似文献
1
Exposure to low level environmental agents: the induction of hormesis.暴露于低水平环境因子：毒物兴奋效应的诱导。
Mutat Res. 2011 Dec 24;726(2):89-90. doi: 10.1016/j.mrgentox.2011.07.008. Epub 2011 Jul 23.
2
An illusion of hormesis in the Ames test: statistical significance is not equivalent to biological significance.Ames 试验中的拟 hormesis 现象：统计学显著性并不等同于生物学显著性。
Mutat Res. 2012 Jul 4;746(1):89-93. doi: 10.1016/j.mrgentox.2012.03.008. Epub 2012 Mar 29.
3
A detailed re-assessment supports the conclusion of the paper that hormesis is commonly observed in the Ames assay.详细的重新评估支持了该论文的结论，即阿米斯试验中普遍观察到毒物兴奋效应。
Mutat Res. 2012 Aug 30;747(1):157. doi: 10.1016/j.mrgentox.2012.04.008. Epub 2012 Apr 28.
4
Evidence for hormesis in mutagenicity dose-response relationships.诱变剂致突变性剂量反应关系中的兴奋效应证据。
Mutat Res. 2011 Dec 24;726(2):91-7. doi: 10.1016/j.mrgentox.2011.04.006. Epub 2011 Apr 21.
5
[Selection and application of mutagenicity tests in identification of carcinogens and mutagens. (II)].
Zhonghua Yu Fang Yi Xue Za Zhi. 1983 Sep;17(5):313-5.
6
Evaluation of Escherichia coli K-12 343/113 and derived strains for microbial mutagenicity assays.
Mutat Res. 1984 Apr;130(2):87-95. doi: 10.1016/0165-1161(84)90108-0.
7
Screening for environmental agents causing genetic damage: introduction.
Lab Pract. 1972 Jun;21(6):411-2.
8
Environmental mutagenesis and carcinogenesis: test method development, validation and utilization.
Mutat Res. 1984 Oct;130(5):353-9. doi: 10.1016/0165-1161(84)90021-9.
9
10
Rapid detection of mutagens accumulated in plant tissues using a novel Vibrio harveyi mutagenicity assay.利用新型哈维氏弧菌致突变性检测法快速检测植物组织中积累的诱变剂。
Ecotoxicol Environ Saf. 2008 Jun;70(2):231-5. doi: 10.1016/j.ecoenv.2007.04.004. Epub 2007 May 18.
引用本文的文献
1
Reduced Fitness and Elevated Oxidative Stress in the Marine Copepod Exposed to the Toxic Dinoflagellate .暴露于有毒甲藻的海洋桡足类动物的健康状况下降及氧化应激增强
Antioxidants (Basel). 2022 Nov 21;11(11):2299. doi: 10.3390/antiox11112299.
2
Projected marine climate change: effects on copepod oxidative status and reproduction.预计的海洋气候变化：对桡足类氧化状态和繁殖的影响。
Ecol Evol. 2013 Nov;3(13):4548-57. doi: 10.1002/ece3.839. Epub 2013 Oct 21.
3
Paradoxical and bidirectional drug effects.矛盾和双向药物效应。
Drug Saf. 2012 Mar 1;35(3):173-89. doi: 10.2165/11597710-000000000-00000.
4
Ionizing radiation-induced metabolic oxidative stress and prolonged cell injury.电离辐射诱导的代谢氧化应激和细胞损伤持续存在。
Cancer Lett. 2012 Dec 31;327(1-2):48-60. doi: 10.1016/j.canlet.2011.12.012. Epub 2011 Dec 17.
本文引用的文献
1
Increased frequency of spontaneous neoplastic transformation in progeny of bystander cells from cultures exposed to densely ionizing radiation.受密集电离辐射照射的细胞旁观者后代中自发性肿瘤转化频率增加。
PLoS One. 2011;6(6):e21540. doi: 10.1371/journal.pone.0021540. Epub 2011 Jun 28.
2
Suppressive effects of continuous low-dose-rate γ irradiation on diabetic nephropathy in type II diabetes mellitus model mice.连续低剂量率γ射线照射对 2 型糖尿病模型小鼠糖尿病肾病的抑制作用。
Radiat Res. 2011 Sep;176(3):356-65. doi: 10.1667/rr2559.1. Epub 2011 Jun 30.
3
Evidence for hormesis in mutagenicity dose-response relationships.诱变剂致突变性剂量反应关系中的兴奋效应证据。
Mutat Res. 2011 Dec 24;726(2):91-7. doi: 10.1016/j.mrgentox.2011.04.006. Epub 2011 Apr 21.
4
The role of gap junction communication and oxidative stress in the propagation of toxic effects among high-dose α-particle-irradiated human cells.缝隙连接通讯和氧化应激在高剂量α粒子辐照的人细胞之间毒性效应传播中的作用。
Radiat Res. 2011 Mar;175(3):347-57. doi: 10.1667/RR2372.1. Epub 2011 Jan 10.
5
Evidence of an adaptive response targeting DNA nonhomologous end joining and its transmission to bystander cells.靶向 DNA 非同源末端连接的适应性反应及其向旁观者细胞传递的证据。
Cancer Res. 2010 Nov 1;70(21):8498-506. doi: 10.1158/0008-5472.CAN-10-1181. Epub 2010 Sep 22.
6
Hormesis is central to toxicology, pharmacology and risk assessment.激效是毒理学、药理学和风险评估的核心。
Hum Exp Toxicol. 2010 Apr;29(4):249-61. doi: 10.1177/0960327109363973.
7
Response of normal stem cells to ionizing radiation: a balance between homeostasis and genomic stability.正常干细胞对电离辐射的反应：维持内稳态和基因组稳定性之间的平衡。
Mutat Res. 2010 Apr-Jun;704(1-3):167-74. doi: 10.1016/j.mrrev.2010.01.007. Epub 2010 Feb 1.
8
Does scientific evidence support a change from the LNT model for low-dose radiation risk extrapolation?科学证据是否支持从低剂量辐射风险外推的线性无阈模型（LNT模型）转变？
Health Phys. 2009 Nov;97(5):493-504. doi: 10.1097/HP.0b013e3181b08a20.
9
Cellular stress response pathway system as a sentinel ensemble in toxicological screening.细胞应激反应通路系统作为毒理学筛查中的一个前哨整体。
Toxicol Sci. 2009 Oct;111(2):202-25. doi: 10.1093/toxsci/kfp140. Epub 2009 Jun 30.
10
Whole-body responses to low-level radiation exposure: new concepts in mammalian radiobiology.对低水平辐射暴露的全身反应：哺乳动物放射生物学的新概念
Exp Hematol. 2007 Apr;35(4 Suppl 1):37-46. doi: 10.1016/j.exphem.2007.01.011.
Zotero 插件