Stefanko Georgia, Lancashire Bill, Coombes Jeff S, Fassett Robert G
Prince of Wales Hospital, Sydney, New South Wales, 2000, Australia.
BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.04.2009.1764. Epub 2009 Aug 17.
A 36-year-old man presented with symptoms of acute pulmonary oedema at the conclusion of the Australian ironman triathlon. He was alert, orientated, with an oxygen saturation of 75% on room air. Chest examination revealed bilateral basal crepitations. Serum sodium was 120 mmol/L and chest x ray revealed bilateral basal opacities. He was treated for acute pulmonary oedema with prompt improvement and given 200 ml of intravenous hypertonic saline followed by normal saline. Serum sodium decreased to 117 mmol/L and 30 hours after presentation he had a seizure. He fully recovered and was discharged 5 days after admission. This case highlights that exercise-associated hyponatraemia and pulmonary oedema are still not widely understood and there is still a reluctance to treat hyponatraemia aggressively with ongoing hypertonic saline.
一名36岁男子在澳大利亚铁人三项赛结束时出现急性肺水肿症状。他意识清醒,定向力正常,室内空气中氧饱和度为75%。胸部检查发现双侧肺底有湿啰音。血清钠为120 mmol/L,胸部X光显示双侧肺底有模糊影。他接受了急性肺水肿治疗,症状迅速改善,并静脉输注了200毫升高渗盐水,随后是生理盐水。血清钠降至117 mmol/L,就诊30小时后他发生了癫痫发作。他完全康复,入院5天后出院。该病例突出表明,运动相关性低钠血症和肺水肿仍未得到广泛认识,对于持续使用高渗盐水积极治疗低钠血症仍存在顾虑。