Is macrophage death on the field of battle essential to victory, or a tactical weakness in immunity against tuberculosis?
作者信息
Lowrie D B
机构信息
National Institute for Medical Research, Mill Hill, London, England.
出版信息
Clin Exp Immunol. 1990 Jun;80(3):301-3. doi: 10.1111/j.1365-2249.1990.tb03285.x.
Abstract
摘要
相似文献
1
Is macrophage death on the field of battle essential to victory, or a tactical weakness in immunity against tuberculosis?巨噬细胞在战斗现场的死亡对于胜利至关重要，还是对抗结核病免疫中的一个战术弱点？
Clin Exp Immunol. 1990 Jun;80(3):301-3. doi: 10.1111/j.1365-2249.1990.tb03285.x.
2
Roles of cytotoxic delayed-type hypersensitivity and macrophage-activating cell-mediated immunity in the pathogenesis of tuberculosis.细胞毒性迟发型超敏反应和巨噬细胞激活的细胞介导免疫在结核病发病机制中的作用。
Immunobiology. 1994 Oct;191(4-5):461-73. doi: 10.1016/S0171-2985(11)80452-3.
3
4
[Recent advances in tuberculosis immunity].[结核病免疫的最新进展]
Kekkaku. 2012 Feb;87(2):61-70.
5
Mycobacterium tuberculosis-Induced Polarization of Human Macrophage Orchestrates the Formation and Development of Tuberculous Granulomas In Vitro.结核分枝杆菌诱导的人巨噬细胞极化在体外调控结核性肉芽肿的形成与发展。
PLoS One. 2015 Jun 19;10(6):e0129744. doi: 10.1371/journal.pone.0129744. eCollection 2015.
6
[Cellular mediated and humoral immunity of tuberculosis].[结核病的细胞介导免疫和体液免疫]
Z Erkr Atmungsorgane. 1982;158(1-2):243-50.
7
Macrophages and Mycobacterium tuberculosis: the key to pathogenesis.巨噬细胞与结核分枝杆菌：发病机制的关键
Immunol Ser. 1994;60:249-61.
8
Mechanisms of immunity in tuberculosis.结核病的免疫机制
Pathobiol Annu. 1979;9:137-62.
9
Virulent clinical isolates of Mycobacterium tuberculosis grow rapidly and induce cellular necrosis but minimal apoptosis in murine macrophages.结核分枝杆菌的强毒临床分离株生长迅速，可诱导小鼠巨噬细胞发生细胞坏死，但凋亡程度极小。
J Leukoc Biol. 2006 Jan;79(1):80-6. doi: 10.1189/jlb.0505250. Epub 2005 Nov 7.
10
[Recent acquisitions on the behavior of macrophages in the acquired immunity against tuberculosis].[关于巨噬细胞在获得性抗结核免疫中行为的最新研究成果]
Arch Monaldi. 1972 May-Jun;27(3):246-71.
引用本文的文献
1
Growth within macrophages increases the efficiency of Mycobacterium avium in invading other macrophages by a complement receptor-independent pathway.巨噬细胞内的生长通过不依赖补体受体的途径提高了鸟分枝杆菌侵入其他巨噬细胞的效率。
Infect Immun. 1997 May;65(5):1916-25. doi: 10.1128/iai.65.5.1916-1925.1997.
2
Depletion of endogenous interleukin-10 augments interleukin-1 beta secretion by Mycobacterium bovis BCG-reactive human cells.内源性白细胞介素-10的耗竭增强了牛分枝杆菌卡介苗反应性人类细胞的白细胞介素-1β分泌。
Clin Diagn Lab Immunol. 1997 Mar;4(2):138-41. doi: 10.1128/cdli.4.2.138-141.1997.
3
Immune responsiveness and lymphokine production in patients with tuberculosis and healthy controls.结核病患者与健康对照者的免疫反应性及淋巴因子产生
Infect Immun. 1994 Dec;62(12):5673-8. doi: 10.1128/iai.62.12.5673-5678.1994.
4
Human Mycobacterium tuberculosis-reactive CD4+ T-cell clones: heterogeneity in antigen recognition, cytokine production, and cytotoxicity for mononuclear phagocytes.人结核分枝杆菌反应性CD4 + T细胞克隆：抗原识别、细胞因子产生及对单核吞噬细胞细胞毒性的异质性
Infect Immun. 1991 Aug;59(8):2737-43. doi: 10.1128/iai.59.8.2737-2743.1991.
5
Unusual spinal tuberculosis after adequate chemotherapy for lymph node tuberculosis in an immunocompetent man.一名免疫功能正常的男性在接受充分化疗治疗淋巴结结核后发生罕见的脊柱结核。
Thorax. 1992 Nov;47(11):986-7. doi: 10.1136/thx.47.11.986.
6
Generation of cytolytic T cells in individuals infected by Mycobacterium tuberculosis and vaccinated with BCG.在感染结核分枝杆菌并接种卡介苗的个体中产生细胞毒性T细胞。
Thorax. 1992 Sep;47(9):695-701. doi: 10.1136/thx.47.9.695.
本文引用的文献
1
Analysis of the host-parasite equilibrium in chronic murine tuberculosis by total and viable bacillary counts.通过总菌数和活菌数计数分析慢性小鼠结核病中的宿主-寄生虫平衡。
Br J Exp Pathol. 1961 Feb;42(1):83-8.
2
Adoptive protection of the Mycobacterium tuberculosis-infected lung. Dissociation between cells that passively transfer protective immunity and those that transfer delayed-type hypersensitivity to tuberculin.结核分枝杆菌感染肺部的过继性保护。被动转移保护性免疫的细胞与转移对结核菌素迟发型超敏反应的细胞之间的分离。
Cell Immunol. 1984 Mar;84(1):113-20. doi: 10.1016/0008-8749(84)90082-0.
3
The relationship of delayed hypersensitivity to acquired antituberculous immunity. II. Effect of adjuvant on the allergenicity and immunogenicity of heat-killed tubercle bacilli.迟发型超敏反应与获得性抗结核免疫的关系。II. 佐剂对热灭活结核杆菌变应原性和免疫原性的影响。
Cell Immunol. 1970 Sep;1(3):266-75. doi: 10.1016/0008-8749(70)90048-1.
4
Gamma interferon activates human macrophages to become tumoricidal and leishmanicidal but enhances replication of macrophage-associated mycobacteria.γ干扰素可激活人类巨噬细胞，使其具有杀肿瘤细胞和杀利什曼原虫的能力，但会增强巨噬细胞相关分枝杆菌的复制。
Infect Immun. 1985 Oct;50(1):1-8. doi: 10.1128/iai.50.1.1-8.1985.
5
I-A restricted activation by T cell lines of anti-tuberculosis activity in murine macrophages.I-A限制小鼠巨噬细胞中抗结核活性的T细胞系激活。
Clin Exp Immunol. 1985 Feb;59(2):414-20.
6
7
CD8+ T lymphocytes in intracellular microbial infections.细胞内微生物感染中的CD8 + T淋巴细胞
Immunol Today. 1988 Jun;9(6):168-74. doi: 10.1016/0167-5699(88)91292-3.
8
Recombinant granulocyte/macrophage colony-stimulating factor activates macrophages to inhibit Trypanosoma cruzi and release hydrogen peroxide. Comparison with interferon gamma.重组粒细胞/巨噬细胞集落刺激因子激活巨噬细胞以抑制克氏锥虫并释放过氧化氢。与干扰素γ的比较。
J Exp Med. 1987 Dec 1;166(6):1734-46. doi: 10.1084/jem.166.6.1734.
9
Recombinant human granulocyte/macrophage colony-stimulating factor activates intracellular killing of Leishmania donovani by human monocyte-derived macrophages.重组人粒细胞/巨噬细胞集落刺激因子激活人单核细胞衍生巨噬细胞对杜氏利什曼原虫的细胞内杀伤作用。
J Exp Med. 1987 Nov 1;166(5):1436-46. doi: 10.1084/jem.166.5.1436.
10
Impaired resistance to Mycobacterium tuberculosis infection after selective in vivo depletion of L3T4+ and Lyt-2+ T cells.体内选择性清除L3T4 +和Lyt-2 + T细胞后，对结核分枝杆菌感染的抵抗力受损。
Infect Immun. 1987 Sep;55(9):2037-41. doi: 10.1128/iai.55.9.2037-2041.1987.
Zotero 插件