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Analysis of subcellular G3BP redistribution during rubella virus infection.
J Gen Virol. 2012 Feb;93(Pt 2):267-274. doi: 10.1099/vir.0.036780-0. Epub 2011 Oct 12.
2
Herpes simplex virus 2 infection impacts stress granule accumulation.
J Virol. 2012 Aug;86(15):8119-30. doi: 10.1128/JVI.00313-12. Epub 2012 May 23.
4
Stress granule components G3BP1 and G3BP2 play a proviral role early in Chikungunya virus replication.
J Virol. 2015 Apr;89(8):4457-69. doi: 10.1128/JVI.03612-14. Epub 2015 Feb 4.
5
Getah virus Nsp3 binds G3BP to block formation of bona fide stress granules.
Int J Biol Macromol. 2024 Nov;279(Pt 2):135274. doi: 10.1016/j.ijbiomac.2024.135274. Epub 2024 Sep 1.
6
Sequestration of G3BP coupled with efficient translation inhibits stress granules in Semliki Forest virus infection.
Mol Biol Cell. 2012 Dec;23(24):4701-12. doi: 10.1091/mbc.E12-08-0619. Epub 2012 Oct 19.
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Formation of antiviral cytoplasmic granules during orthopoxvirus infection.
J Virol. 2011 Feb;85(4):1581-93. doi: 10.1128/JVI.02247-10. Epub 2010 Dec 8.
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Inhibition of cytoplasmic mRNA stress granule formation by a viral proteinase.
Cell Host Microbe. 2007 Nov 15;2(5):295-305. doi: 10.1016/j.chom.2007.08.006.

引用本文的文献

1
Two Birds With One Stone: RNA Virus Strategies to Manipulate G3BP1 and Other Stress Granule Components.
Wiley Interdiscip Rev RNA. 2025 Mar-Apr;16(2):e70005. doi: 10.1002/wrna.70005.
2
Copy-back viral genomes induce a cellular stress response that interferes with viral protein expression without affecting antiviral immunity.
PLoS Biol. 2023 Nov 20;21(11):e3002381. doi: 10.1371/journal.pbio.3002381. eCollection 2023 Nov.
3
Role(s) of G3BPs in Human Pathogenesis.
J Pharmacol Exp Ther. 2023 Oct;387(1):100-110. doi: 10.1124/jpet.122.001538. Epub 2023 Jul 19.
4
Stress granules: potential therapeutic targets for infectious and inflammatory diseases.
Front Immunol. 2023 May 2;14:1145346. doi: 10.3389/fimmu.2023.1145346. eCollection 2023.
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Role of Stress Granules in Suppressing Viral Replication by the Infectious Bronchitis Virus Endoribonuclease.
J Virol. 2022 Jun 22;96(12):e0068622. doi: 10.1128/jvi.00686-22. Epub 2022 May 31.
6
Research Progress on the Structure and Function of G3BP.
Front Immunol. 2021 Aug 30;12:718548. doi: 10.3389/fimmu.2021.718548. eCollection 2021.
7
Infectious Bronchitis Virus Regulates Cellular Stress Granule Signaling.
Viruses. 2020 May 14;12(5):536. doi: 10.3390/v12050536.
8
Strategies for Success. Viral Infections and Membraneless Organelles.
Front Cell Infect Microbiol. 2019 Oct 11;9:336. doi: 10.3389/fcimb.2019.00336. eCollection 2019.
9
Regulation of Stress Granule Formation by Inflammation, Vascular Injury, and Atherosclerosis.
Arterioscler Thromb Vasc Biol. 2019 Oct;39(10):2014-2027. doi: 10.1161/ATVBAHA.119.313034. Epub 2019 Aug 29.
10

本文引用的文献

1
Analysis of the function of cytoplasmic fibers formed by the rubella virus nonstructural replicase proteins.
Virology. 2010 Oct 25;406(2):212-27. doi: 10.1016/j.virol.2010.07.025. Epub 2010 Aug 8.
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Determinants of subcellular localization of the rubella virus nonstructural replicase proteins.
Virology. 2009 Aug 1;390(2):315-23. doi: 10.1016/j.virol.2009.05.019. Epub 2009 Jun 18.
5
Different types of nsP3-containing protein complexes in Sindbis virus-infected cells.
J Virol. 2008 Oct;82(20):10088-101. doi: 10.1128/JVI.01011-08. Epub 2008 Aug 6.
6
P bodies, stress granules, and viral life cycles.
Cell Host Microbe. 2008 Apr 17;3(4):206-12. doi: 10.1016/j.chom.2008.03.004.
7
Rubella virus capsid protein interacts with poly(a)-binding protein and inhibits translation.
J Virol. 2008 May;82(9):4284-94. doi: 10.1128/JVI.02732-07. Epub 2008 Feb 27.
8
Inhibition of cytoplasmic mRNA stress granule formation by a viral proteinase.
Cell Host Microbe. 2007 Nov 15;2(5):295-305. doi: 10.1016/j.chom.2007.08.006.

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