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Activity-dependent increase in neurotrophic factors is associated with an enhanced modulation of spinal reflexes after spinal cord injury.
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Intramuscular AAV delivery of NT-3 alters synaptic transmission to motoneurons in adult rats.
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Cycling exercise affects the expression of apoptosis-associated microRNAs after spinal cord injury in rats.
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PTEN deletion enhances the regenerative ability of adult corticospinal neurons.
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Mammalian target of rapamycin (mTOR) activation increases axonal growth capacity of injured peripheral nerves.
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PTEN/mTOR and axon regeneration.
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Proprioceptive neuropathy affects normalization of the H-reflex by exercise after spinal cord injury.
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Altered microRNA expression following traumatic spinal cord injury.
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