Synaptic activity in X-linked mental retardation: a thorny issue.
作者信息
Martina Marco
机构信息
Department of Physiology, Northwestern University, Feinberg School of Medicine, 300 E Chicago Avenue, Chicago, IL 60611, USA.
出版信息
J Physiol. 2012 Feb 15;590(4):653. doi: 10.1113/jphysiol.2011.226019.
Abstract
摘要
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本文引用的文献
1
Rapid reversal of impaired inhibitory and excitatory transmission but not spine dysgenesis in a mouse model of mental retardation.智力迟钝小鼠模型中，抑制和兴奋传递受损的快速逆转，但不是脊柱发育不良。
J Physiol. 2012 Feb 15;590(4):763-76. doi: 10.1113/jphysiol.2011.219907. Epub 2011 Nov 28.
2
Dendritic spines and distributed circuits.树突棘和分布式电路。
Neuron. 2011 Sep 8;71(5):772-81. doi: 10.1016/j.neuron.2011.07.024.
3
Loss of X-linked mental retardation gene oligophrenin1 in mice impairs spatial memory and leads to ventricular enlargement and dendritic spine immaturity.小鼠中X连锁智力迟钝基因少突磷脂1的缺失会损害空间记忆，并导致脑室扩大和树突棘发育不成熟。
J Neurosci. 2007 Aug 29;27(35):9439-50. doi: 10.1523/JNEUROSCI.2029-07.2007.
4
The X-linked mental retardation protein oligophrenin-1 is required for dendritic spine morphogenesis.X连锁智力迟钝蛋白少突神经胶质细胞1是树突棘形态发生所必需的。
Nat Neurosci. 2004 Apr;7(4):364-72. doi: 10.1038/nn1210. Epub 2004 Mar 14.
5
Calcium microdomains in aspiny dendrites.无棘树突中的钙微区
Neuron. 2003 Nov 13;40(4):807-21. doi: 10.1016/s0896-6273(03)00714-1.
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