Goicolea de Oro A, Chicote R, López-Gil M, Márquez L, Botella M, García-Cosío F
Servicio de Cardiología, Hospital Central de la Cruz Roja, Madrid.
Rev Esp Cardiol. 1990;43 Suppl 2:52-7.
A consecutive series of 18 patients (5 males, 13 females, mean age +/- DS 65 +/- 12 and 66 +/- 13 years, respectively, and mean +/- DS Wenckebach point of 162 +/- 20 ppm) given arterial pacemakers for sinus node dysfunction (SND) were followed to study the incidence of lead failure, atrioventricular conduction disturbances and chronic atrial tachyarrhythmias. The mean follow up time +/- SD was 18 +/- 10 months. There was not lead dislodgement. Chronic voltage output was reduced to 2.5 volts in 73% of patients because of a reduced chronic pacing threshold. One patient presented acute transient rise of pacing threshold and temporary loss of atrial detection. Progression to AV block was not documented. One patient had asymptomatic Wenckebach AV block during the night while on digoxin plus amiodarone. The AV block disappeared after cessation of drug therapy. Patients with previous episodes of paroxysmal atrial fibrillation did not presented the arrhythmia during follow up, while 75% of patients with previous atrial flutter presented the arrhythmia. None of the patients had systemic embolism. In conclusion, AAI pacing is a reliable and safe mode of pacing in patients with SND.
连续选取18例因窦房结功能障碍（SND）植入动脉起搏器的患者（5例男性，13例女性，平均年龄±标准差分别为65±12岁和66±13岁，平均±标准差文氏点为162±20 ppm），对其进行随访，以研究导线故障、房室传导障碍和慢性房性快速性心律失常的发生率。平均随访时间±标准差为18±10个月。未发生导线移位。由于慢性起搏阈值降低，73%的患者慢性电压输出降至2.5伏。1例患者出现起搏阈值急性短暂升高和心房感知暂时丧失。未记录到进展为房室传导阻滞的情况。1例患者在服用地高辛加胺碘酮期间夜间出现无症状文氏型房室传导阻滞。停药后房室传导阻滞消失。既往有阵发性心房颤动发作的患者在随访期间未出现心律失常，而既往有心房扑动的患者中有75%出现了心律失常。所有患者均未发生系统性栓塞。总之，AAI起搏是SND患者可靠且安全的起搏模式。
