National Center for PTSD at VA BostonHealthcare System, Boston, Massachusetts 02130, USA.
Personal Disord. 2011 Oct;2(4):261-78. doi: 10.1037/a0023168.
Research on the structure of personality disorders (PDs) has relied primarily on exploratory analyses to evaluate trait-based models of the factors underlying the covariation of these disorders. This study used confirmatory factor analysis to evaluate whether a model that included both PD traits and a general personality dysfunction factor would account for the comorbidity of the PDs better than a trait-only model. It also examined if the internalizing/externalizing model of psychopathology, developed previously through research on the structure of Axis I disorders, might similarly account for the covariation of the Axis II disorders in a sample of 245 veterans and nonveterans with posttraumatic stress disorder. Results indicated that the best fitting model was a modified bifactor structure composed of nine lower-order common factors. These factors indexed pathology ranging from aggression to dependency, with the correlations among them accounted for by higher-order Internalizing and Externalizing factors. Further, a general factor, reflecting a construct that we termed boundary disturbance, accounted for additional variance and covariance across nearly all the indicators. The Internalizing, Externalizing, and Boundary Disturbance factors evidenced differential associations with trauma-related covariates. These findings suggest continuity in the underlying structure of psychopathology across DSM-IV Axes I and II and provide empirical evidence of a pervasive, core disturbance in the boundary between self and other across the PDs.
人格障碍(PDs)结构的研究主要依赖于探索性分析,以评估潜在这些障碍共变的特质模型。本研究采用验证性因子分析来评估包括 PD 特质和一般人格功能障碍因素的模型是否比仅基于特质的模型更能解释 PD 的共病现象。它还检查了先前通过轴 I 障碍结构研究开发的心理病理学的内外向模型是否也可以同样解释创伤后应激障碍患者样本中轴 II 障碍的共变。结果表明,最合适的模型是由九个低阶共同因素组成的改良双因素结构。这些因素反映了从攻击性到依赖性的病理学,它们之间的相关性由高阶的内外向因素来解释。此外,一个反映我们称之为边界障碍的建构的一般因素,解释了几乎所有指标的额外差异和协方差。内外向和边界障碍因素与与创伤相关的协变量有不同的关联。这些发现表明,DSM-IV 轴 I 和 II 之间的心理病理学潜在结构具有连续性,并提供了一个普遍的、核心的边界障碍的实证证据,跨越了 PDs。