• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

噬菌体中裂解抑制的筛选。

Selection for lysis inhibition in bacteriophage.

作者信息

Abedon S T

机构信息

Department of Microbiology and Immunology, University of Arizona, Tucson 85724.

出版信息

J Theor Biol. 1990 Oct 21;146(4):501-11. doi: 10.1016/s0022-5193(05)80375-3.

DOI:10.1016/s0022-5193(05)80375-3
PMID:2273898
Abstract

For Escherichia coli cells that have been infected by T-even bacteriophages (phages T2, T4, and T6), the adsorption of a second T-even phage results in an increase in the length of the original phage infection and an associated increase in the number of phages produced by the same infected cell. This is a phage encoded response called lysis inhibition. In this study the ecological significance of lysis inhibition is explored. In particular it is argued that lysis inhibition is an adaptive response to environments containing high concentrations of infected cells and low concentrations of uninfected cells.

摘要

对于已被T偶数噬菌体(噬菌体T2、T4和T6)感染的大肠杆菌细胞,第二个T偶数噬菌体的吸附会导致原始噬菌体感染时间延长,且同一被感染细胞产生的噬菌体数量相应增加。这是一种由噬菌体编码的反应,称为裂解抑制。在本研究中,探讨了裂解抑制的生态学意义。特别是有人认为,裂解抑制是对含有高浓度感染细胞和低浓度未感染细胞的环境的一种适应性反应。

相似文献

1
Selection for lysis inhibition in bacteriophage.噬菌体中裂解抑制的筛选。
J Theor Biol. 1990 Oct 21;146(4):501-11. doi: 10.1016/s0022-5193(05)80375-3.
2
Lysis of lysis-inhibited bacteriophage T4-infected cells.溶菌抑制性噬菌体T4感染细胞的裂解
J Bacteriol. 1992 Dec;174(24):8073-80. doi: 10.1128/jb.174.24.8073-8080.1992.
3
Competitive advantages of T-even phage lysis inhibition in response to secondary infection.T-偶数噬菌体溶菌抑制对二次感染的竞争优势。
PLoS Comput Biol. 2024 Jul 8;20(7):e1012242. doi: 10.1371/journal.pcbi.1012242. eCollection 2024 Jul.
4
Roles of bacteriophage T4 gene 5 and gene s products in cell lysis.噬菌体T4基因5和基因s产物在细胞裂解中的作用。
J Virol. 1980 Apr;34(1):104-7. doi: 10.1128/JVI.34.1.104-107.1980.
5
Look Who's Talking: T-Even Phage Lysis Inhibition, the Granddaddy of Virus-Virus Intercellular Communication Research.看谁在说话:T偶数噬菌体裂解抑制,病毒-病毒细胞间通讯研究的鼻祖。
Viruses. 2019 Oct 16;11(10):951. doi: 10.3390/v11100951.
6
Baseplate protein of bacteriophage T4 with both structural and lytic functions.噬菌体T4的基板蛋白,兼具结构和裂解功能。
J Virol. 1980 Apr;34(1):95-103. doi: 10.1128/JVI.34.1.95-103.1980.
7
Interrupted thymidylate synthase gene of bacteriophages T2 and T6 and other potential self-splicing introns in the T-even bacteriophages.噬菌体T2和T6的胸苷酸合成酶基因中断以及T偶数噬菌体中的其他潜在自我剪接内含子。
J Bacteriol. 1987 Sep;169(9):4368-75. doi: 10.1128/jb.169.9.4368-4375.1987.
8
Mechanism of cell wall penetration by viruses. II. Demonstration of cyclic permeability change accompanying virus infection of Escherichia coli B cells.病毒穿透细胞壁的机制。II. 大肠杆菌B细胞受病毒感染时伴随的循环通透性变化的证明。
J Exp Med. 1955 Feb 1;101(2):151-75. doi: 10.1084/jem.101.2.151.
9
[Effect of low temperatures of the survival and intracellular multiplication of Escherichia coli bacteriophages].[低温对大肠杆菌噬菌体存活及细胞内增殖的影响]
Mikrobiologiia. 1981 Mar-Apr;50(2):292-4.
10
The action of Escherichia coli CRISPR-Cas system on lytic bacteriophages with different lifestyles and development strategies.大肠杆菌CRISPR-Cas系统对具有不同生活方式和发育策略的裂解性噬菌体的作用。
Nucleic Acids Res. 2017 Feb 28;45(4):1946-1957. doi: 10.1093/nar/gkx042.

引用本文的文献

1
Lysis Physiology of Infected with ssRNA Phage PRR1.感染单链RNA噬菌体PRR1后的裂解生理学
Viruses. 2024 Apr 21;16(4):645. doi: 10.3390/v16040645.
2
Potential of an Isolated Bacteriophage to Inactivate : Preliminary Studies to Control Urinary Tract Infections.一种分离出的噬菌体使[细菌]失活的潜力:控制尿路感染的初步研究
Antibiotics (Basel). 2024 Feb 19;13(2):195. doi: 10.3390/antibiotics13020195.
3
How Simple Maths Can Inform Our Basic Understanding of Phage Therapy.简单数学如何帮助我们基本理解噬菌体疗法。
Clin Infect Dis. 2023 Nov 2;77(Suppl 5):S401-S406. doi: 10.1093/cid/ciad480.
4
Further Considerations on How to Improve Phage Therapy Experimentation, Practice, and Reporting: Pharmacodynamics Perspectives.关于如何改进噬菌体治疗实验、实践及报告的进一步思考:药效学视角
Phage (New Rochelle). 2022 Jun 1;3(2):98-111. doi: 10.1089/phage.2022.0019. Epub 2022 Jun 16.
5
Repeated outbreaks drive the evolution of bacteriophage communication.噬菌体的反复爆发驱动了其通讯方式的进化。
Elife. 2021 Jan 18;10:e58410. doi: 10.7554/eLife.58410.
6
Dominant phage exhibits lysis inhibition sensitive to disruption by a defensive phage satellite.优势噬菌体表现出对防御性噬菌体卫星破坏敏感的裂解抑制。
Elife. 2020 Apr 24;9:e53200. doi: 10.7554/eLife.53200.
7
Linking Light-Dependent Life History Traits with Population Dynamics for and Cyanophage.将光依赖性生活史特征与蓝细菌和噬蓝藻体的种群动态联系起来。
mSystems. 2020 Mar 31;5(2):e00586-19. doi: 10.1128/mSystems.00586-19.
8
Look Who's Talking: T-Even Phage Lysis Inhibition, the Granddaddy of Virus-Virus Intercellular Communication Research.看谁在说话:T偶数噬菌体裂解抑制,病毒-病毒细胞间通讯研究的鼻祖。
Viruses. 2019 Oct 16;11(10):951. doi: 10.3390/v11100951.
9
Commentary: A Host-Produced Quorum-Sensing Autoinducer Controls a Phage Lysis-Lysogeny Decision.评论:宿主产生的群体感应自诱导物控制噬菌体的裂解-溶原决定。
Front Microbiol. 2019 Jun 3;10:1171. doi: 10.3389/fmicb.2019.01171. eCollection 2019.
10
Commentary: Communication between Viruses Guides Lysis-Lysogeny Decisions.述评:病毒间的通讯指导裂解-溶原决定。
Front Microbiol. 2017 May 31;8:983. doi: 10.3389/fmicb.2017.00983. eCollection 2017.