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KCNJ10 mutations disrupt function in patients with EAST syndrome.KCNJ10 突变会破坏 EAST 综合征患者的功能。
Nephron Physiol. 2011;119(3):p40-8. doi: 10.1159/000330250. Epub 2011 Aug 18.
2
Transient receptor potential channels as therapeutic targets.瞬时受体电位通道作为治疗靶点。
Nat Rev Drug Discov. 2011 Aug 1;10(8):601-20. doi: 10.1038/nrd3456.
3
Role of BK channels in hypertension and potassium secretion.BK 通道在高血压和钾分泌中的作用。
Curr Opin Nephrol Hypertens. 2011 Sep;20(5):512-7. doi: 10.1097/MNH.0b013e3283488889.
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Aurora A kinase activity influences calcium signaling in kidney cells.极光激酶 A 的活性会影响肾脏细胞中的钙信号转导。
J Cell Biol. 2011 Jun 13;193(6):1021-32. doi: 10.1083/jcb.201012061.
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Clinical and molecular characterization of Turkish patients with familial hypomagnesaemia: novel mutations in TRPM6 and CLDN16 genes.土耳其家族性低镁血症患者的临床和分子特征：TRPM6 和 CLDN16 基因的新突变。
Nephrol Dial Transplant. 2012 Feb;27(2):667-73. doi: 10.1093/ndt/gfr300. Epub 2011 Jun 9.
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Structural model of the TRPP2/PKD1 C-terminal coiled-coil complex produced by a combined computational and experimental approach.采用计算与实验相结合的方法构建 TRPP2/PKD1 C 端卷曲螺旋复合物的结构模型。
Proc Natl Acad Sci U S A. 2011 Jun 21;108(25):10133-8. doi: 10.1073/pnas.1017669108. Epub 2011 Jun 3.
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Macromolecular assembly of polycystin-2 intracytosolic C-terminal domain.多囊蛋白-2胞质内环 C 端结构域的高分子组装。
Proc Natl Acad Sci U S A. 2011 Jun 14;108(24):9833-8. doi: 10.1073/pnas.1106766108. Epub 2011 May 27.
8
Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia.醛固酮诱导的遗传性低镁血症模型中，瞬时受体电位 melastatin 6/7 的肾脏调节紊乱，但原钙黏蛋白-1 无变化，导致高血压和肾脏损害。
J Hypertens. 2011 Jul;29(7):1400-10. doi: 10.1097/HJH.0b013e32834786d6.
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Generation and analyses of R8L barttin knockin mouse.R8L barttin 敲入鼠的产生和分析。
Am J Physiol Renal Physiol. 2011 Aug;301(2):F297-307. doi: 10.1152/ajprenal.00604.2010. Epub 2011 May 18.
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Sodium-dependent phosphate cotransporters: lessons from gene knockout and mutation studies.钠依赖性磷酸盐协同转运蛋白：基因敲除和突变研究的启示。
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Ion channels in renal disease.

作者信息

Kuo Ivana Y, Ehrlich Barbara E

机构信息

Departments of †Pharmacology and ‡Cellular and Molecular Physiology School of Medicine, Yale University , 333 Cedar Street, New Haven, Connecticut 06520.

出版信息

Chem Rev. 2012 Dec 12;112(12):6353-72. doi: 10.1021/cr3001077. Epub 2012 Jul 18.

DOI:10.1021/cr3001077
PMID:22809040
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3511917/
Abstract
摘要