Knox B E, Gates G A, Berry S M
Department of Otolaryngology, University of Texas Health Science Center, San Antonio.
Laryngoscope. 1990 May;100(5):458-62. doi: 10.1288/00005537-199005000-00003.
Two cases of visual loss after lateral orbital wall fracture are presented: one with retrobulbar hematoma and evidence of optic nerve compression who failed to respond to lateral canthotomy and high-dose corticosteroid administration, and the second with immediate, total blindness associated with fracture of the bony optic canal. In both, extradural decompression of the orbit and optic nerve was achieved through the lateral facial approach with partial return of visual acuity and without surgical complications. The role of orbital and optic nerve decompression in the management of patients with blindness following orbital trauma is controversial. Orbital decompression may be of value for cases of post-traumatic visual loss unresponsive to medical management. If optic nerve injury is suspected as the cause, the additional step of decompression of the optic nerve is a logical but unproven procedure. The indications for optic nerve decompression are not established and should be considered only within the context of the specific needs of the individual patient.
一例伴有球后血肿及视神经受压证据,经外眦切开术和大剂量皮质类固醇治疗无效;另一例伴有骨性视神经管骨折,伤后立即完全失明。两例均通过面外侧入路实现了眶和视神经的硬膜外减压,视力部分恢复,且无手术并发症。眶减压和视神经减压在眼眶外伤致盲患者治疗中的作用存在争议。眶减压对于药物治疗无效的创伤后视力丧失病例可能有价值。如果怀疑视神经损伤是病因,额外进行视神经减压是合理但未经证实的操作。视神经减压的指征尚未确立,应仅在个体患者的特定需求背景下考虑。