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本文引用的文献

1
Prenatal bisphenol a exposure and child behavior in an inner-city cohort.产前双酚 A 暴露与城市内城区队列儿童行为。
Environ Health Perspect. 2012 Aug;120(8):1190-4. doi: 10.1289/ehp.1104492. Epub 2012 Apr 27.
2
Quantitation of free and total bisphenol A in human urine using liquid chromatography-tandem mass spectrometry.采用液相色谱-串联质谱法定量测定人尿液中的游离和总双酚 A。
J Sep Sci. 2011 Jun;34(11):1268-74. doi: 10.1002/jssc.201100087. Epub 2011 Apr 20.
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Determination of free and total bisphenol A in urine of infants.测定婴幼儿尿液中的游离和总双酚 A。
Environ Res. 2011 Jan;111(1):143-8. doi: 10.1016/j.envres.2010.10.001.
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Determination of bisphenol A in U.S. infant formulas: updated methods and concentrations.测定美国婴儿配方食品中的双酚 A:方法和浓度更新。
J Agric Food Chem. 2010 Feb 24;58(4):2307-13. doi: 10.1021/jf903959u.
5
Predicting plasma concentrations of bisphenol A in children younger than 2 years of age after typical feeding schedules, using a physiologically based toxicokinetic model.使用基于生理学的毒代动力学模型预测2岁以下儿童在典型喂养时间表后的双酚A血浆浓度。
Environ Health Perspect. 2009 Apr;117(4):645-52. doi: 10.1289/ehp.0800073. Epub 2008 Nov 14.
6
Exposure to bisphenol A and other phenols in neonatal intensive care unit premature infants.新生儿重症监护病房早产儿接触双酚A和其他酚类物质的情况。
Environ Health Perspect. 2009 Apr;117(4):639-44. doi: 10.1289/ehp.0800265. Epub 2008 Dec 10.
7
Human exposure to bisphenol A by biomonitoring: methods, results and assessment of environmental exposures.通过生物监测评估人类对双酚A的暴露:方法、结果及环境暴露评估
Toxicol Appl Pharmacol. 2008 Apr 1;228(1):114-34. doi: 10.1016/j.taap.2007.12.008. Epub 2007 Dec 14.
8
Exposure of the U.S. population to bisphenol A and 4-tertiary-octylphenol: 2003-2004.2003 - 2004年美国人群对双酚A和4 - 叔辛基苯酚的暴露情况。
Environ Health Perspect. 2008 Jan;116(1):39-44. doi: 10.1289/ehp.10753.
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Pharmacokinetics in the newborn.新生儿的药代动力学
Adv Drug Deliv Rev. 2003 Apr 29;55(5):667-86. doi: 10.1016/s0169-409x(03)00030-9.
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Metabolism and kinetics of bisphenol a in humans at low doses following oral administration.口服低剂量双酚A后在人体中的代谢及动力学
Chem Res Toxicol. 2002 Oct;15(10):1281-7. doi: 10.1021/tx025548t.

新生儿尿中游离双酚 A 和双酚 A-葡糖苷酸浓度。

Urinary free bisphenol A and bisphenol A-glucuronide concentrations in newborns.

机构信息

Department of Environmental Health Sciences, Johns Hopkins Bloomberg School of Public Health, Baltimore, MD 21205, USA.

出版信息

J Pediatr. 2013 Apr;162(4):870-2. doi: 10.1016/j.jpeds.2012.11.083. Epub 2013 Jan 11.

DOI:10.1016/j.jpeds.2012.11.083
PMID:23312693
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3609880/
Abstract

Infants are exposed to the endocrine disruptor bisphenol A (BPA) through breast milk and baby formula. Detoxication by conjugation of BPA may be limited in infants. We demonstrate BPA exposure in 11 neonates and 1 young infant, but find no evidence of a low capacity for BPA conjugation.

摘要

婴儿通过母乳和婴儿配方奶粉接触到内分泌干扰物双酚 A(BPA)。BPA 的共轭解毒可能在婴儿中受到限制。我们在 11 名新生儿和 1 名幼儿中证实了 BPA 的暴露,但没有发现 BPA 共轭能力低下的证据。