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The adaptor MAVS promotes NLRP3 mitochondrial localization and inflammasome activation.
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Succinate is an inflammatory signal that induces IL-1β through HIF-1α.
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TRPM2 links oxidative stress to NLRP3 inflammasome activation.
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Microtubule-driven spatial arrangement of mitochondria promotes activation of the NLRP3 inflammasome.
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Noncanonical inflammasomes: caspase-11 activation and effector mechanisms.
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The protein kinase PKR is critical for LPS-induced iNOS production but dispensable for inflammasome activation in macrophages.
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Caspase-11 protects against bacteria that escape the vacuole.
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Deubiquitination of NLRP3 by BRCC3 critically regulates inflammasome activity.
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