Mouse alopecia areata and heart disease: know your mouse!
作者信息
Sundberg John P, Chevallier Lucie, Silva Kathleen A, Kennedy Victoria E, Sundberg Beth A, Li Qiaoli, Uitto Jouni, King Lloyd E, Berndt Annerose
机构信息
The Jackson Laboratory, Bar Harbor, Maine, USA; Division of Dermatology, Department of Medicine, Vanderbilt University, Nashville, Tennessee, USA.
The Jackson Laboratory, Bar Harbor, Maine, USA.
出版信息
J Invest Dermatol. 2014 Jan;134(1):279-281. doi: 10.1038/jid.2013.273. Epub 2013 Jun 17.
Abstract
摘要
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Mouse alopecia areata and heart disease: know your mouse!小鼠斑秃与心脏病：了解你的小鼠！
J Invest Dermatol. 2014 Jan;134(1):279-281. doi: 10.1038/jid.2013.273. Epub 2013 Jun 17.
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Major locus on mouse chromosome 17 and minor locus on chromosome 9 are linked with alopecia areata in C3H/HeJ mice.在C3H/HeJ小鼠中，位于17号染色体上的主要基因座和位于9号染色体上的次要基因座与斑秃有关。
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C3H/HeJ mouse model for alopecia areata.斑秃的C3H/HeJ小鼠模型
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Spontaneous alopecia areata-like hair loss in one congenic and seven inbred laboratory mouse strains.一种近交系和七种近交实验室小鼠品系中出现的自发性斑秃样脱发。
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Increased expression of Cxcr3 and its ligands, Cxcl9 and Cxcl10, during the development of alopecia areata in the mouse.在斑秃小鼠模型的发病过程中，趋化因子受体3（Cxcr3）及其配体Cxcl9和Cxcl10的表达增加。
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[Alopecia areata in animal models--new insights into pathogenesis and treatment of a T cell-mediated autoimmune disorder].[动物模型中的斑秃——对一种T细胞介导的自身免疫性疾病发病机制及治疗的新见解]
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Behaviour-associated alopecia areata in mice.小鼠行为相关的斑秃
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Fas-deficient C3.MRL-Tnfrsf6(lpr) mice and Fas ligand-deficient C3H/HeJ-Tnfsf6(gld) mice are relatively resistant to the induction of alopecia areata by grafting of alopecia areata-affected skin from C3H/HeJ mice.Fas缺陷的C3.MRL-Tnfrsf6(lpr)小鼠和Fas配体缺陷的C3H/HeJ-Tnfsf6(gld)小鼠，通过移植来自C3H/HeJ小鼠的斑秃病变皮肤，相对不易被诱导发生斑秃。
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Adult-onset Alopecia areata is a complex polygenic trait in the C3H/HeJ mouse model.在C3H/HeJ小鼠模型中，成年期斑秃是一种复杂的多基因性状。
J Invest Dermatol. 2004 Aug;123(2):294-7. doi: 10.1111/j.0022-202X.2004.23222.x.
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Alopecia areata in aging C3H/HeJ mice.衰老C3H/HeJ小鼠中的斑秃
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本文引用的文献
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A single-nucleotide polymorphism in the Abcc6 gene associates with connective tissue mineralization in mice similar to targeted models for pseudoxanthoma elasticum.Abcc6基因中的单核苷酸多态性与小鼠的结缔组织矿化有关，类似于弹性假黄瘤的靶向模型。
J Invest Dermatol. 2013 Mar;133(3):833-836. doi: 10.1038/jid.2012.340. Epub 2012 Sep 27.
2
The mouse as a model for understanding chronic diseases of aging: the histopathologic basis of aging in inbred mice.小鼠作为理解衰老相关慢性疾病的模型：近交系小鼠衰老的组织病理学基础。
Pathobiol Aging Age Relat Dis. 2011;1. doi: 10.3402/pba.v1i0.7179. Epub 2011 Jun 1.
3
Alopecia areata as a model for T cell-dependent autoimmune diseases.斑秃作为T细胞依赖性自身免疫性疾病的模型。
Exp Dermatol. 2012 Jun;21(6):477-9. doi: 10.1111/j.1600-0625.2011.01427.x.
4
Pseudoxanthoma elasticum: molecular genetics and putative pathomechanisms.弹性假黄瘤：分子遗传学和可能的发病机制。
J Invest Dermatol. 2010 Mar;130(3):661-70. doi: 10.1038/jid.2009.411. Epub 2009 Dec 24.
5
Dysferlin deficiency and the development of cardiomyopathy in a mouse model of limb-girdle muscular dystrophy 2B.肢带型肌营养不良症 2B 模型小鼠的 dysferlin 缺乏与心肌病的发展。
Am J Pathol. 2009 Dec;175(6):2299-308. doi: 10.2353/ajpath.2009.080930. Epub 2009 Oct 29.
6
An alternative splice variant in Abcc6, the gene causing dystrophic calcification, leads to protein deficiency in C3H/He mice.导致营养不良性钙化的基因Abcc6中的一种可变剪接变体，会导致C3H/He小鼠出现蛋白质缺乏。
J Biol Chem. 2008 Mar 21;283(12):7608-15. doi: 10.1074/jbc.M708290200. Epub 2008 Jan 16.
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Identification of Abcc6 as the major causal gene for dystrophic cardiac calcification in mice through integrative genomics.通过整合基因组学鉴定Abcc6为小鼠营养不良性心脏钙化的主要致病基因。
Proc Natl Acad Sci U S A. 2007 Mar 13;104(11):4530-5. doi: 10.1073/pnas.0607620104. Epub 2007 Mar 6.
8
Targeted ablation of the abcc6 gene results in ectopic mineralization of connective tissues.ABCC6基因的靶向消融导致结缔组织的异位矿化。
Mol Cell Biol. 2005 Sep;25(18):8299-310. doi: 10.1128/MCB.25.18.8299-8310.2005.
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Disruption of Abcc6 in the mouse: novel insight in the pathogenesis of pseudoxanthoma elasticum.小鼠中Abcc6基因的破坏：弹性假黄瘤发病机制的新见解。
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Physiol Genomics. 2001 Aug 28;6(3):137-44. doi: 10.1152/physiolgenomics.2001.6.3.137.
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