Himeno Takahiro, Takeshima Shinnichi, Kubo Satoshi, Hara Naoyuki, Takamatsu Kazuhiro, Kuriyama Masaru
Department of Neurology, Brain Attack Center Ota Memorial Hospital.
Rinsho Shinkeigaku. 2013;53(6):478-81. doi: 10.5692/clinicalneurol.53.478.
A 39-year-old man was suffered from bacterial meningitis spread from sphenoid sinusitis. During the first several days of the hospitalization, his clinical and laboratory findings were improved by the antibiotics. But he developed impaired consciousness and paraparesis on the sixth hospital day. A CT scan of the brain revealed pneumocephalus with compression of frontal lobes and the widening of the interhemispheric space between the tips of the frontal lobes, which was known as "Mount Fuji sign". Tension pneumocephalus was diagnosed on the basis of the clinical symptoms and the characteristic CT findings. As the bacterial meningitis itself was improving, the surgical treatment was not performed, but the antibiotics therapy continued. He gradually recovered and discharged without any other complications. The mechanism of tension pneumocephalus could not be disclosed. However, it was speculated that tension pneumocephalus was formed due to combined conditions of following factors; the fistula formation between sphenoid sinus and subdural space, the reduced CSF pressure on lumbar puncture, and a ball-valve mechanism though the fistula. We would emphasize that "Mount Fuji sign"on CT or MRI was the important finding to diagnose tension pneumocephalus.
一名39岁男性因蝶窦炎引发细菌性脑膜炎。在住院的最初几天,抗生素治疗使他的临床和实验室检查结果有所改善。但在住院第6天,他出现意识障碍和双下肢轻瘫。脑部CT扫描显示存在气颅,额叶受压,额叶尖端之间的半球间间隙增宽,即所谓的“富士山征”。根据临床症状和特征性CT表现诊断为张力性气颅。由于细菌性脑膜炎本身正在好转,未进行手术治疗,但继续使用抗生素治疗。他逐渐康复并出院,无任何其他并发症。张力性气颅的机制尚不清楚。然而,推测张力性气颅是由于以下因素共同作用形成的:蝶窦与硬膜下间隙之间形成瘘管、腰椎穿刺后脑脊液压力降低以及通过瘘管的球阀机制。我们强调CT或MRI上的“富士山征”是诊断张力性气颅的重要发现。