Torrente D, Avila M F, Cabezas R, Morales L, Gonzalez J, Samudio I, Barreto G E
Departamento de Nutrición y Bioquímica, Facultad de Ciencias, Pontificia Universidad Javeriana, Bogotá DC, Colombia.
Departamento de Nutrición y Bioquímica, Facultad de Ciencias, Pontificia Universidad Javeriana, Bogotá DC, Colombia
Hum Exp Toxicol. 2014 Jul;33(7):673-84. doi: 10.1177/0960327113509659. Epub 2013 Oct 31.
Traumatic brain injury (TBI) consists of a primary and a secondary insult characterized by a biochemical cascade that plays a crucial role in cell death in the brain. Despite the major improvements in the acute care of head injury victims, no effective strategies exist for preventing the secondary injury cascade. This lack of success might be due to that most treatments are aimed at targeting neuronal population, even if studies show that astrocytes play a key role after a brain damage. In this work, we propose a new model of in vitro traumatic brain-like injury and use paracrine factors released by human mesenchymal stem cells (hMSCs) as a neuroprotective strategy. Our results demonstrate that hMSC-conditioned medium increased wound closure and proliferation at 12 h and reduced superoxide production to control conditions. This was accompanied by changes in cell morphology and polarity index, as both parameters reflect the ability of cells to migrate toward the wound. These findings indicate that hMSC is an important regulator of oxidative stress production, enhances cells migration, and shall be considered as a useful neuroprotective approach for brain recovery following injury.
创伤性脑损伤(TBI)由原发性损伤和继发性损伤组成,其特征是存在一个在脑内细胞死亡中起关键作用的生化级联反应。尽管头部受伤患者的急性护理有了重大改善,但目前尚无有效的策略来预防继发性损伤级联反应。这种缺乏成效的情况可能是由于大多数治疗方法都针对神经元群体,即便研究表明星形胶质细胞在脑损伤后发挥关键作用。在这项研究中,我们提出了一种体外创伤性脑样损伤的新模型,并将人间充质干细胞(hMSCs)释放的旁分泌因子用作一种神经保护策略。我们的结果表明,hMSC条件培养基在12小时时增加了伤口闭合和细胞增殖,并将超氧化物产生量降低至对照水平。这伴随着细胞形态和极性指数的变化,因为这两个参数都反映了细胞向伤口迁移的能力。这些发现表明,hMSC是氧化应激产生的重要调节因子,可增强细胞迁移,应被视为损伤后脑恢复的一种有用的神经保护方法。
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