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本文引用的文献
1
Synergistic regulation of hyphal elongation by hypoxia, CO(2), and nutrient conditions controls the virulence of Candida albicans.
Cell Host Microbe. 2013 Nov 13;14(5):499-509. doi: 10.1016/j.chom.2013.10.008.
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Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes.
Eukaryot Cell. 2013 Jan;12(1):37-49. doi: 10.1128/EC.00236-12. Epub 2012 Nov 2.
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A GATA transcription factor recruits Hda1 in response to reduced Tor1 signaling to establish a hyphal chromatin state in Candida albicans.
PLoS Pathog. 2012;8(4):e1002663. doi: 10.1371/journal.ppat.1002663. Epub 2012 Apr 19.
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Hypoxia and fungal pathogenesis: to air or not to air?
Eukaryot Cell. 2012 May;11(5):560-70. doi: 10.1128/EC.00031-12. Epub 2012 Mar 23.
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Growth of Candida albicans hyphae.
Nat Rev Microbiol. 2011 Aug 16;9(10):737-48. doi: 10.1038/nrmicro2636.
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Hyphal development in Candida albicans requires two temporally linked changes in promoter chromatin for initiation and maintenance.
PLoS Biol. 2011 Jul;9(7):e1001105. doi: 10.1371/journal.pbio.1001105. Epub 2011 Jul 19.
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Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity.
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Sensing the environment: response of Candida albicans to the X factor.
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UME6, a novel filament-specific regulator of Candida albicans hyphal extension and virulence.
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