Mechanisms of sterile inflammation.
作者信息
Rubartelli Anna, Lotze Michael T, Latz Eicke, Manfredi Angelo
机构信息
Cell Biology, IRCCS AOU San Martino IST , Genova , Italy.
出版信息
Front Immunol. 2013 Nov 22;4:398. doi: 10.3389/fimmu.2013.00398. eCollection 2013.
Abstract
摘要
相似文献
1
Mechanisms of sterile inflammation.无菌性炎症的机制。
Front Immunol. 2013 Nov 22;4:398. doi: 10.3389/fimmu.2013.00398. eCollection 2013.
2
NLRP3-Inflammasome Activating DAMPs Stimulate an Inflammatory Response in Glia in the Absence of Priming Which Contributes to Brain Inflammation after Injury.NLRP3炎性小体激活的内源性危险信号分子在未启动的情况下刺激神经胶质细胞产生炎症反应，这在损伤后导致脑炎症。
Front Immunol. 2012 Sep 18;3:288. doi: 10.3389/fimmu.2012.00288. eCollection 2012.
3
Role of the Nalp3 inflammasome in acetaminophen-induced sterile inflammation and liver injury.NALP3 炎性体在对乙酰氨基酚诱导的无菌性炎症和肝损伤中的作用。
Toxicol Appl Pharmacol. 2011 May 1;252(3):289-97. doi: 10.1016/j.taap.2011.03.001. Epub 2011 Mar 21.
4
DAMPs as mediators of sterile inflammation in aging-related pathologies.危险相关分子模式作为与衰老相关病理学中无菌性炎症的介质。
Ageing Res Rev. 2015 Nov;24(Pt A):29-39. doi: 10.1016/j.arr.2015.01.003. Epub 2015 Jan 29.
5
How is inflammation initiated? Individual influences of IL-1, IL-18 and HMGB1.炎症是如何引发的？白细胞介素-1、白细胞介素-18和高迁移率族蛋白B1的个体影响。
Cytokine. 2014 Sep;69(1):136-45. doi: 10.1016/j.cyto.2014.03.007. Epub 2014 Apr 16.
6
DAMPs and sterile inflammation in drug hepatotoxicity.药物性肝毒性中的 DAMPs 和无菌性炎症。
Hepatol Int. 2019 Jan;13(1):42-50. doi: 10.1007/s12072-018-9911-9. Epub 2018 Nov 24.
7
Gefitinib initiates sterile inflammation by promoting IL-1β and HMGB1 release via two distinct mechanisms.吉非替尼通过两种不同的机制促进 IL-1β 和 HMGB1 的释放，引发无菌性炎症。
Cell Death Dis. 2021 Jan 6;12(1):49. doi: 10.1038/s41419-020-03335-7.
8
Danger-Associated Molecular Patterns (DAMPs): the Derivatives and Triggers of Inflammation.危险相关分子模式（DAMPs）：炎症的衍生物和触发物。
Curr Allergy Asthma Rep. 2018 Sep 28;18(11):63. doi: 10.1007/s11882-018-0817-3.
9
The inflammasome and danger associated molecular patterns (DAMPs) are implicated in cytokine and chemokine responses following stressor exposure.炎症小体和相关的危险相关分子模式（DAMPs）参与应激暴露后的细胞因子和趋化因子反应。
Brain Behav Immun. 2013 Feb;28:54-62. doi: 10.1016/j.bbi.2012.10.014. Epub 2012 Oct 24.
10
Etiological Value of Sterile Inflammation in Preeclampsia: Is It a Non-Infectious Pregnancy Complication?子痫前期无菌性炎症的病因学价值：它是一种非感染性妊娠并发症吗？
Front Cell Infect Microbiol. 2021 Aug 16;11:694298. doi: 10.3389/fcimb.2021.694298. eCollection 2021.
引用本文的文献
1
Genetic line-specific immune profiles and immunometabolic responses to intramuscular lipopolysaccharide injection.特定遗传系谱的免疫谱以及对肌肉注射脂多糖的免疫代谢反应。
Front Immunol. 2025 Jun 16;16:1608391. doi: 10.3389/fimmu.2025.1608391. eCollection 2025.
2
CeO@ CuO nanoparticle alleviates inflammatory responses by regulating the CTSB-NLRP3 signaling pathway.CeO@CuO 纳米颗粒通过调节 CTSB-NLRP3 信号通路缓解炎症反应。
Front Immunol. 2024 Apr 22;15:1344098. doi: 10.3389/fimmu.2024.1344098. eCollection 2024.
3
Non-Neuronal Acetylcholinesterase Activity Shows Limited Utility for Early Detection of Sepsis.非神经元乙酰胆碱酯酶活性在脓毒症早期检测中的应用价值有限。
Biomedicines. 2023 Jul 26;11(8):2111. doi: 10.3390/biomedicines11082111.
4
Obstructive nephropathy and molecular pathophysiology of renal interstitial fibrosis.梗阻性肾病与肾间质纤维化的分子病理生理学
Physiol Rev. 2023 Oct 1;103(4):2827-2872. doi: 10.1152/physrev.00027.2022. Epub 2023 Jul 13.
5
The Molecular Basis and Clinical Consequences of Chronic Inflammation in Prostatic Diseases: Prostatitis, Benign Prostatic Hyperplasia, and Prostate Cancer.前列腺疾病中慢性炎症的分子基础及临床后果：前列腺炎、良性前列腺增生和前列腺癌。
Cancers (Basel). 2023 Jun 8;15(12):3110. doi: 10.3390/cancers15123110.
6
P2Y/IL-1 receptor crosstalk controls macrophage inflammation: a novel target for anti-inflammatory strategies?P2Y/IL-1 受体相互作用控制巨噬细胞炎症：抗炎策略的新靶点？
Purinergic Signal. 2023 Sep;19(3):501-511. doi: 10.1007/s11302-023-09932-3. Epub 2023 Apr 5.
7
Putative Role of Neutrophil Extracellular Trap Formation in Chronic Myeloproliferative Neoplasms.中性粒细胞胞外诱捕网形成在慢性骨髓增殖性肿瘤中的推测作用。
Int J Mol Sci. 2023 Feb 24;24(5):4497. doi: 10.3390/ijms24054497.
8
The role of macrophage subtypes and exosomes in immunomodulation.巨噬细胞亚型和外泌体在免疫调节中的作用。
Cell Mol Biol Lett. 2022 Oct 3;27(1):83. doi: 10.1186/s11658-022-00384-y.
9
The P2Y receptor of human M2 macrophages activates canonical and IL-1 receptor signaling to translate the extracellular danger signal ATP into anti-inflammatory and pro-angiogenic responses.人源 M2 巨噬细胞的 P2Y 受体激活经典和 IL-1 受体信号通路，将细胞外危险信号 ATP 转化为抗炎和促血管生成反应。
Cell Mol Life Sci. 2022 Sep 15;79(10):519. doi: 10.1007/s00018-022-04548-z.
10
Biodistribution of intravenous [Tc]Tc-phytate in mouse models of chemically and foreign-body induced sterile inflammation.静脉注射[锝]锝-植酸盐在化学诱导和异物诱导的无菌性炎症小鼠模型中的生物分布。
Am J Nucl Med Mol Imaging. 2022 Jun 15;12(3):91-98. eCollection 2022.
本文引用的文献
1
HMGB1: The Central Cytokine for All Lymphoid Cells.高迁移率族蛋白 B1：所有淋巴样细胞的核心细胞因子。
Front Immunol. 2013 Mar 20;4:68. doi: 10.3389/fimmu.2013.00068. eCollection 2013.
2
Mechanisms of disease: inflammasome activation and the development of type 2 diabetes.发病机制：炎性体激活与 2 型糖尿病的发生。
Front Immunol. 2013 Mar 8;4:50. doi: 10.3389/fimmu.2013.00050. eCollection 2013.
3
Endometriosis, a disease of the macrophage.子宫内膜异位症，一种巨噬细胞疾病。
Front Immunol. 2013 Jan 28;4:9. doi: 10.3389/fimmu.2013.00009. eCollection 2013.
4
ATP release and purinergic signaling in NLRP3 inflammasome activation.三磷酸腺苷释放和嘌呤能信号在 NLRP3 炎性小体激活中的作用。
Front Immunol. 2013 Jan 8;3:414. doi: 10.3389/fimmu.2012.00414. eCollection 2012.
5
TIR8/SIGIRR is an Interleukin-1 Receptor/Toll Like Receptor Family Member with Regulatory Functions in Inflammation and Immunity.TIR8/SIGIRR 是白细胞介素-1 受体/ Toll 样受体家族成员，在炎症和免疫中具有调节功能。
Front Immunol. 2012 Oct 29;3:322. doi: 10.3389/fimmu.2012.00322. eCollection 2012.
6
The entry of fetal and amniotic fluid components into the uterine vessel circulation leads to sterile inflammatory processes during parturition.胎儿和羊水成分进入子宫血管循环会导致分娩过程中的无菌性炎症过程。
Front Immunol. 2012 Oct 23;3:321. doi: 10.3389/fimmu.2012.00321. eCollection 2012.
7
IL-1 family cytokines trigger sterile inflammatory disease.白细胞介素-1 家族细胞因子引发无菌性炎症性疾病。
Front Immunol. 2012 Oct 9;3:315. doi: 10.3389/fimmu.2012.00315. eCollection 2012.
8
The transcription of the alarmin cytokine interleukin-1 alpha is controlled by hypoxia inducible factors 1 and 2 alpha in hypoxic cells.在缺氧细胞中，警报素细胞因子白细胞介素-1α的转录受缺氧诱导因子 1 和 2α的控制。
Front Immunol. 2012 Sep 14;3:290. doi: 10.3389/fimmu.2012.00290. eCollection 2012.
9
NLRP3-Inflammasome Activating DAMPs Stimulate an Inflammatory Response in Glia in the Absence of Priming Which Contributes to Brain Inflammation after Injury.NLRP3炎性小体激活的内源性危险信号分子在未启动的情况下刺激神经胶质细胞产生炎症反应，这在损伤后导致脑炎症。
Front Immunol. 2012 Sep 18;3:288. doi: 10.3389/fimmu.2012.00288. eCollection 2012.
10
DAMP signaling in fungal infections and diseases.真菌病感染和疾病中的 damp 信号。
Front Immunol. 2012 Sep 10;3:286. doi: 10.3389/fimmu.2012.00286. eCollection 2012.
Zotero 插件