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眼眶减压术对甲状腺相关性眼病复视的影响。

Effect of orbital decompression on diplopia in thyroid-related orbitopathy.

机构信息

*Department of Ophthalmology, Sudbury Regional Hospital, Sudbury; and †Department of Ophthalmology, University of Ottawa Eye Institute and The Ottawa Hospital, Ottawa, Ontario, Canada.

出版信息

Ophthalmic Plast Reconstr Surg. 2014 Mar-Apr;30(2):137-40. doi: 10.1097/IOP.0000000000000029.

Abstract

PURPOSE

To determine the incidence of new-onset diplopia and evolution of preexisting diplopia in patients with thyroid-related orbitopathy undergoing orbital decompression surgery.

METHODS

A retrospective chart review was conducted of patients who had undergone orbital decompression for thyroid-related orbitopathy between 1999 and 2008 in one of the authors' practice (D.J.). A total of 217 orbits in 123 patients were identified. The clinical indication for decompression surgery (i.e., exposure keratitis, optic neuropathy, or improvement of cosmesis) was recorded in each case, as was the presence of pre- and postoperative diplopia. The surgical technique (1-, 2-, or 3-wall decompression) was noted for each patient.

RESULTS

Review of the charts of patients who underwent orbital decompression surgery for thyroid-related orbitopathy revealed a preoperative prevalence of diplopia of 26% and a postoperative prevalence of 40.7%. Amongst the patients with preoperative diplopia (n = 32), 28.1% (n = 9) had complete resolution of their diplopia after decompression, while 65.6% (n = 21) remained stable and 6.3% (n = 2) worsened. The incidence of new-onset diplopia was 29.7% in this case series of orbital decompression using a transcaruncular and swinging eyelid approach for medial wall and strut-sparing floor decompression. Rates of new-onset diplopia were significantly higher when periorbita was opened (40.0%, n = 82) compared with when it was left intact (11.8%, n = 37) CONCLUSIONS:: It has previously been reported in the literature that orbital decompression for thyroid-related orbitopathy can cause diplopia in a significant number of cases. This provides the rational for performing orbital decompression prior to strabismus surgery in the management of thyroid-related orbitopathy. In this case series, the authors noted resolution of diplopia in a significant proportion (28.1%) of patients with preexisting diplopia. This is rarely commented on in other articles but is important in the preoperative discussion. An incidence of new-onset diplopia of 29.7% was identified. Opening the periorbita was associated with an increased incidence of new-onset diplopia.

摘要

目的

确定甲状腺相关眼病行眼眶减压术后新发复视和原有复视的演变情况。

方法

对 1999 年至 2008 年间在作者之一(D.J.)所在机构接受甲状腺相关眼病眼眶减压术的患者进行回顾性图表分析。共确定了 123 例患者的 217 只眼。记录了每种情况下减压手术的临床适应证(即暴露性角膜炎、视神经病变或美容改善),以及术前和术后复视的存在情况。注意到每位患者的手术技术(1-、2-或 3-壁减压)。

结果

回顾甲状腺相关眼病行眼眶减压术患者的图表显示,术前复视患病率为 26%,术后患病率为 40.7%。在术前有复视的患者中(n=32),28.1%(n=9)减压后复视完全缓解,65.6%(n=21)稳定,6.3%(n=2)恶化。在本眼眶减压病例系列中,经结膜和摆动眼睑入路进行内侧壁和支柱保留的眶底减压,新发复视的发生率为 29.7%。打开眶周组织(40.0%,n=82)与保持眶周组织完整(11.8%,n=37)相比,新发性复视的发生率显著更高。

结论

文献中已有报道称,甲状腺相关眼病的眼眶减压术可导致相当数量的病例出现复视。这为甲状腺相关眼病的眼眶减压术在斜视手术之前进行提供了合理的依据。在本病例系列中,作者注意到有相当比例(28.1%)的原有复视患者复视得到缓解。这在其他文章中很少被提及,但在术前讨论中很重要。新发性复视的发生率为 29.7%。打开眶周组织与新发复视的发生率增加有关。

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