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艰难梭菌复发的特征是促炎性外周血单核细胞(PBMC)表型。

Clostridium difficile recurrence is characterized by pro-inflammatory peripheral blood mononuclear cell (PBMC) phenotype.

作者信息

Yacyshyn Mary B, Reddy Tara N, Plageman Lauren R, Wu Jiang, Hollar Amy R, Yacyshyn Bruce R

机构信息

Division of Digestive Diseases, Department of Medicine, University of Cincinnati, Cincinnati, Ohio, USA.

Department of Internal Medicine, University of Cincinnati, Cincinnati, Ohio, USA.

出版信息

J Med Microbiol. 2014 Oct;63(Pt 10):1260-1273. doi: 10.1099/jmm.0.075382-0. Epub 2014 Jul 7.

Abstract

Clostridium difficile infection (CDI) is a prevalent nosocomial and increasingly community-acquired problem. Little is known about the productive cellular response in patients. We used flow cytometry to define inflammatory (Th1 and Th17) and regulatory [Foxp3(+) T-regulatory (Treg)] cells present in circulating peripheral blood mononuclear cells (PBMC) from CDI patients. We consented 67 inpatients that tested either positive or negative for CDI and 16 healthy controls and compared their PBMC phenotypes. PBMC were collected, isolated, and stained for CD3, CD8 and either IL17 (Th17), IFN-γ (Th1) or Foxp3 (Treg) and analysed using flow cytometry. Twenty thousand events were collected in the lymphocyte gate (gate 1) and T-cell phenotypes were defined. CDI patients who clear the primary initial infection have greater numbers of non-CD3 PBMC. CDI patients who develop recurrence of CDI have a greater percentage of CD3(+)CD8(+), CD3(+)CD4(+)Foxp3 and fewer low granular CD3(-)Foxp3(+) PBMC. These patients have greater numbers of IFN-γ-producing lymphocytes, as well as PBMC phenotypes represented by increased IFN-γ- and IL17-co-expressing CD4(+)CD3(+). This initial pro-inflammatory phenotype decreases with repeated recurrence, demonstrating importance of timing of sample collection and history of symptoms. Patients with a history of recurrence had increased Foxp3(+)CD3(+)CD4(+) and IL17(+)CD3(+)CD4(+) populations. Hence, CDI recurrence is hallmarked by greater numbers of circulating CD3(+) lymphocytes skewed towards a Th1/Th17 inflammatory population as well as possible immune plasticity (Th17/Treg).

摘要

艰难梭菌感染(CDI)是一个普遍存在的医院感染问题,且越来越多地出现在社区获得性感染中。对于患者体内有效的细胞反应,我们了解甚少。我们运用流式细胞术来确定存在于CDI患者循环外周血单个核细胞(PBMC)中的炎性细胞(Th1和Th17)及调节性细胞[Foxp3(+) T调节性细胞(Treg)]。我们招募了67名CDI检测呈阳性或阴性的住院患者以及16名健康对照者,并比较了他们的PBMC表型。采集PBMC,进行分离,并用抗CD3、CD8以及IL17(Th17)、IFN-γ(Th1)或Foxp3(Treg)进行染色,然后使用流式细胞术进行分析。在淋巴细胞门(门1)中收集20000个事件,并确定T细胞表型。清除原发性初始感染的CDI患者具有更多数量的非CD3 PBMC。发生CDI复发的患者中,CD3(+)CD8(+)、CD3(+)CD4(+)Foxp3的比例更高,而低颗粒度CD3(-)Foxp3(+) PBMC数量更少。这些患者产生IFN-γ的淋巴细胞数量更多,同时具有以IFN-γ和IL17共表达增加为特征的PBMC表型的CD4(+)CD3(+)。这种初始的促炎表型会随着复发次数的增加而降低,这表明样本采集时间和症状史的重要性。有复发史的患者中,Foxp3(+)CD3(+)CD4(+)和IL17(+)CD3(+)CD4(+)群体增加。因此,CDI复发的特征是循环CD3(+)淋巴细胞数量增加,且偏向Th1/Th17炎性群体,以及可能存在的免疫可塑性(Th17/Treg)。

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