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Antral Helicobacter pylori, hypergastrinaemia, and duodenal ulcers: effect of eradicating the organism.

作者信息

Levi S, Beardshall K, Swift I, Foulkes W, Playford R, Ghosh P, Calam J

机构信息

Department of Medicine, Royal Postgraduate Medical School, London.

出版信息

BMJ. 1989 Dec 16;299(6714):1504-5. doi: 10.1136/bmj.299.6714.1504.

DOI:10.1136/bmj.299.6714.1504
PMID:2514864
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1838341/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/87e0/1838341/610e8d7e256a/bmj00263-0037-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/87e0/1838341/610e8d7e256a/bmj00263-0037-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/87e0/1838341/610e8d7e256a/bmj00263-0037-a.jpg

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Duodenal ulcer, Campylobacter pylori, and the "leaking roof" concept.十二指肠溃疡、幽门螺杆菌与“漏水屋顶”概念
Lancet. 1988;2(8626-8627):1467-9. doi: 10.1016/s0140-6736(88)90942-7.
2
Campylobacter pylori and duodenal ulcers: the gastrin link.
Lancet. 1989 May 27;1(8648):1167-8. doi: 10.1016/s0140-6736(89)92752-9.
3
pH dependence of acid secretion and gastrin release in normal and ulcer subjects.正常人和溃疡患者胃酸分泌及胃泌素释放的pH依赖性
J Clin Invest. 1975 Mar;55(3):462-8. doi: 10.1172/JCI107952.
尼莫地尔体外抗幽门螺杆菌活性的研究。
J Ethnopharmacol. 2022 Mar 1;285:114828. doi: 10.1016/j.jep.2021.114828. Epub 2021 Nov 9.
4
New antituberculous drugs derived from natural products: current perspectives and issues in antituberculous drug development.源自天然产物的新型抗结核药物：抗结核药物研发的现状与问题
J Antibiot (Tokyo). 2017 Nov 1. doi: 10.1038/ja.2017.126.
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Reduced secretion of epidermal growth factor in duodenal ulcer patients with Helicobacter pylori infection.幽门螺杆菌感染的十二指肠溃疡患者表皮生长因子分泌减少。
World J Gastroenterol. 1997 Mar 15;3(1):31-4. doi: 10.3748/wjg.v3.i1.31.
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Helicobacter pylori infection, gastrin and cyclooxygenase-2 in gastric carcinogenesis.幽门螺杆菌感染、胃泌素与环氧化酶-2在胃癌发生中的作用
World J Gastroenterol. 2014 Sep 28;20(36):12860-73. doi: 10.3748/wjg.v20.i36.12860.
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Human gastrin mRNA expression up-regulated by Helicobacter pylori CagA through MEK/ERK and JAK2-signaling pathways in gastric cancer cells.人胃泌素 mRNA 表达受幽门螺杆菌 CagA 通过 MEK/ERK 和 JAK2 信号通路在胃癌细胞中上调。
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