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缺血再灌注损伤

Ischemia-reperfusion damage.

作者信息

Yapca Omer E, Borekci Bunyamin, Suleyman Halis

机构信息

Department of Obstetrics and Gynecology, Sorgun State Hospital, Yozgat, Turkey.

Department of Obstetrics and Gynecology, Ataturk University Faculty of Medicine, Erzurum, Turkey.

出版信息

Eurasian J Med. 2013 Jun;45(2):126-7. doi: 10.5152/eajm.2013.24.

Abstract

Ischemia-reperfusion damage is a complex pathological process that begins with tissue anoxia and continues with the production of free oxygen radicals, expanding with the inflammatory response. The literature suggests the importance of antioxidant and anti-inflammatory treatment to treat ischemia-reperfusion-related tissue damage.

摘要

缺血再灌注损伤是一个复杂的病理过程,始于组织缺氧,接着是氧自由基的产生,并随着炎症反应而扩展。文献表明抗氧化和抗炎治疗对于治疗缺血再灌注相关组织损伤具有重要意义。

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Ischemia-reperfusion damage.缺血再灌注损伤
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引用本文的文献

本文引用的文献

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Tissue damage and oxidant/antioxidant balance.组织损伤与氧化/抗氧化平衡。
Eurasian J Med. 2013 Feb;45(1):47-9. doi: 10.5152/eajm.2013.08.
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Reactive species mechanisms of cellular hypoxia-reoxygenation injury.细胞缺氧-复氧损伤的活性物质机制。
Am J Physiol Cell Physiol. 2002 Feb;282(2):C227-41. doi: 10.1152/ajpcell.00112.2001.
4
Oxyradicals and DNA damage.氧自由基与DNA损伤。
Carcinogenesis. 2000 Mar;21(3):361-70. doi: 10.1093/carcin/21.3.361.
5
Pathophysiology of ischaemia-reperfusion injury.缺血再灌注损伤的病理生理学
J Pathol. 2000 Feb;190(3):255-66. doi: 10.1002/(SICI)1096-9896(200002)190:3<255::AID-PATH526>3.0.CO;2-6.
6
Cellular and molecular dissection of reperfusion injury: ROS within and without.
Circ Res. 2000 Feb 4;86(2):117-8. doi: 10.1161/01.res.86.2.117.
8
Arachidonic acid metabolites in CSF in hypoxic-ischaemic encephalopathy of newborn infants.
Acta Paediatr. 1998 May;87(5):588-92. doi: 10.1080/08035259850158344.
9
Reactive oxygen metabolites, neutrophils, and the pathogenesis of ischemic-tissue/reperfusion.
Clin Cardiol. 1993 Apr;16(4 Suppl 1):I19-26. doi: 10.1002/clc.4960161307.
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The time sequence of injury and recovery following transient reversible intestinal ischemia.
J Surg Res. 1994 Mar;56(3):221-5. doi: 10.1006/jsre.1994.1035.

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