Bodini G, Fiamminghi L
Dent Cadmos. 1989 Mar 31;57(5):68-77.
Latest results about etipatogenesis of oral papillomas make us to modify the old classification according which papillomatosis were traumatic or idiopathic, giving to the latest a tested viral origin. Studies at the electron microscopy, immunoperoxidase technique and DNA-hybridization showed that human papillomavirus (HPV) is the etiologic agent of these oral lesions. Many works seem to support the opinion that HPV could be implicated even in the development of some oral carcionomas; anyway, further confirmatory evidence is still needed.
关于口腔乳头状瘤发病机制的最新研究结果使我们对旧的分类进行了修改，根据旧分类，乳头状瘤病分为创伤性或特发性，而现在认为后者有经证实的病毒起源。电子显微镜、免疫过氧化物酶技术和DNA杂交研究表明，人乳头瘤病毒（HPV）是这些口腔病变的病原体。许多研究似乎支持HPV甚至可能与某些口腔癌的发生有关的观点；无论如何，仍需要进一步的确证证据。
