• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

A Perspective on Chromosomal Double Strand Break Markers in Mammalian Cells.

作者信息

Wang Haibo, Adhikari Sanjay, Butler Brian E, Pandita Tej K, Mitra Sankar, Hegde Muralidhar L

机构信息

Department of Radiation Oncology, Houston Methodist Research Institute, USA.

Houston Methodist Neurological Institute, USA.

出版信息

Jacobs J Radiat Oncol. 2014;1(1).

PMID:25614903
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4299656/
Abstract
摘要

相似文献

1
A Perspective on Chromosomal Double Strand Break Markers in Mammalian Cells.哺乳动物细胞中染色体双链断裂标记物的研究视角
Jacobs J Radiat Oncol. 2014;1(1).
2
DNA double-strand breaks, chromosomal rearrangements, and genomic instability.
Mutat Res. 1998 Aug 3;404(1-2):125-8. doi: 10.1016/s0027-5107(98)00104-3.
3
Enzymatic restriction of mammalian cell DNA using Pvu II and Bam H1: evidence for the double-strand break origin of chromosomal aberrations.使用Pvu II和Bam H1对哺乳动物细胞DNA进行酶切限制:染色体畸变双链断裂起源的证据。
Int J Radiat Biol Relat Stud Phys Chem Med. 1984 Jul;46(1):57-65. doi: 10.1080/09553008414551061.
4
Evidence that extrachromosomal double-strand break repair can be coupled to the repair of chromosomal double-strand breaks in mammalian cells.有证据表明,在哺乳动物细胞中,染色体外双链断裂修复可与染色体双链断裂修复相偶联。
Chromosoma. 2002 Dec;111(5):304-12. doi: 10.1007/s00412-002-0212-6. Epub 2002 Sep 18.
5
Intra-chromosomal gene conversion induced by a DNA double-strand break in Saccharomyces cerevisiae.酿酒酵母中DNA双链断裂诱导的染色体内基因转换。
J Mol Biol. 1988 May 20;201(2):247-60. doi: 10.1016/0022-2836(88)90136-2.
6
Resection and repair of a Cas9 double-strand break at CTG trinucleotide repeats induces local and extensive chromosomal deletions.在 CTG 三核苷酸重复序列处对 Cas9 双链断裂进行切除和修复会诱导局部和广泛的染色体缺失。
PLoS Genet. 2020 Jul 16;16(7):e1008924. doi: 10.1371/journal.pgen.1008924. eCollection 2020 Jul.
7
The rejoining of DNA double-strand breaks and a model for the formation of chromosomal rearrangements.DNA双链断裂的重新连接及染色体重排形成模型。
Int J Radiat Biol Relat Stud Phys Chem Med. 1978 Jun;33(6):517-29. doi: 10.1080/09553007814550431.
8
Involvement of double-strand chromosomal breaks for mating-type switching in Saccharomyces cerevisiae.双链染色体断裂参与酿酒酵母的交配型转换。
Cold Spring Harb Symp Quant Biol. 1984;49:77-88. doi: 10.1101/sqb.1984.049.01.011.
9
DNA double strand break repair and chromosomal translocation: lessons from animal models.DNA双链断裂修复与染色体易位:来自动物模型的经验教训
Oncogene. 2001 Sep 10;20(40):5572-9. doi: 10.1038/sj.onc.1204767.
10
Conservative repair of a chromosomal double-strand break by single-strand DNA through two steps of annealing.通过单链DNA经两步退火对染色体双链断裂进行保守修复。
Mol Cell Biol. 2006 Oct;26(20):7645-57. doi: 10.1128/MCB.00672-06. Epub 2006 Aug 14.

引用本文的文献

1
The Proteasome Inhibitor CEP-18770 Induces Cell Death in Medulloblastoma.蛋白酶体抑制剂CEP-18770诱导髓母细胞瘤细胞死亡。
Pharmaceutics. 2024 May 16;16(5):672. doi: 10.3390/pharmaceutics16050672.
2
DNA Double Strand Break and Response Fluorescent Assays: Choices and Interpretation.DNA 双链断裂和反应荧光分析:选择和解读。
Int J Mol Sci. 2024 Feb 13;25(4):2227. doi: 10.3390/ijms25042227.
3
Cooling of Cells and Organs Confers Extensive DNA Strand Breaks Through Oxidative Stress and ATP Depletion.细胞和器官的冷却通过氧化应激和 ATP 耗竭导致广泛的 DNA 链断裂。
Cell Transplant. 2022 Jan-Dec;31:9636897221108705. doi: 10.1177/09636897221108705.
4
DNA Damage and Repair Deficiency in ALS/FTD-Associated Neurodegeneration: From Molecular Mechanisms to Therapeutic Implication.肌萎缩侧索硬化症/额颞叶痴呆相关神经退行性变中的DNA损伤与修复缺陷:从分子机制到治疗意义
Front Mol Neurosci. 2021 Dec 16;14:784361. doi: 10.3389/fnmol.2021.784361. eCollection 2021.
5
Inter and intra-tumor somatostatin receptor 2 heterogeneity influences peptide receptor radionuclide therapy response.肿瘤内和肿瘤间生长抑素受体 2 异质性影响肽受体放射性核素治疗反应。
Theranostics. 2021 Jan 1;11(2):491-505. doi: 10.7150/thno.51215. eCollection 2021.
6
Radiation-induced DNA double-strand breaks in peripheral leukocytes and therapeutic response of heel spur patients treated by orthovoltage X-rays or a linear accelerator.外周血白细胞辐射诱导的 DNA 双链断裂与Orthovoltage X 射线或线性加速器治疗跟骨刺患者的治疗反应。
Strahlenther Onkol. 2020 Dec;196(12):1116-1127. doi: 10.1007/s00066-020-01662-4. Epub 2020 Jul 10.
7
Connexin43 Hemichannel Targeting With TAT-Gap19 Alleviates Radiation-Induced Endothelial Cell Damage.利用TAT-Gap19靶向连接蛋白43半通道可减轻辐射诱导的内皮细胞损伤。
Front Pharmacol. 2020 Mar 5;11:212. doi: 10.3389/fphar.2020.00212. eCollection 2020.
8
Pervasive Genomic Damage in Experimental Intracerebral Hemorrhage: Therapeutic Potential of a Mechanistic-Based Carbon Nanoparticle.实验性脑出血中的普遍基因组损伤:基于机制的碳纳米粒子的治疗潜力。
ACS Nano. 2020 Mar 24;14(3):2827-2846. doi: 10.1021/acsnano.9b05821. Epub 2020 Feb 21.
9
Increased double strand breaks in diabetic β-cells with a p21 response that limits apoptosis.糖尿病β细胞中 p21 反应导致的双链断裂增加,从而限制了细胞凋亡。
Sci Rep. 2019 Dec 18;9(1):19341. doi: 10.1038/s41598-019-54554-8.
10
SPOP regulates the DNA damage response and lung adenocarcinoma cell response to radiation.SPOP调节DNA损伤反应以及肺腺癌细胞对辐射的反应。
Am J Cancer Res. 2019 Jul 1;9(7):1469-1483. eCollection 2019.

本文引用的文献

1
New perspectives on oxidized genome damage and repair inhibition by pro-oxidant metals in neurological diseases.神经疾病中氧化基因组损伤及促氧化金属对修复抑制作用的新观点
Biomolecules. 2014 Jul 17;4(3):678-703. doi: 10.3390/biom4030678.
2
MOF phosphorylation by ATM regulates 53BP1-mediated double-strand break repair pathway choice.ATM介导的MOF磷酸化调控53BP1介导的双链断裂修复途径选择。
Cell Rep. 2014 Jul 10;8(1):177-89. doi: 10.1016/j.celrep.2014.05.044. Epub 2014 Jun 19.
3
Role of 53BP1 in the regulation of DNA double-strand break repair pathway choice.53BP1 在调控 DNA 双链断裂修复途径选择中的作用。
Radiat Res. 2014 Jan;181(1):1-8. doi: 10.1667/RR13572.1. Epub 2013 Dec 9.
4
Prereplicative repair of oxidized bases in the human genome is mediated by NEIL1 DNA glycosylase together with replication proteins.人类基因组中氧化碱基的复制前修复是由 NEIL1 DNA 糖苷酶与复制蛋白共同介导的。
Proc Natl Acad Sci U S A. 2013 Aug 13;110(33):E3090-9. doi: 10.1073/pnas.1304231110. Epub 2013 Jul 29.
5
Reduced contribution of thermally labile sugar lesions to DNA double strand break formation after exposure to heavy ions.重离子照射后,热不稳定糖基损伤对 DNA 双链断裂形成的贡献减少。
Radiat Oncol. 2013 Apr 2;8:77. doi: 10.1186/1748-717X-8-77.
6
Chromatin modifications and the DNA damage response to ionizing radiation.染色质修饰与电离辐射的 DNA 损伤反应。
Front Oncol. 2013 Jan 22;2:214. doi: 10.3389/fonc.2012.00214. eCollection 2012.
7
Oxidative genome damage and its repair in neurodegenerative diseases: function of transition metals as a double-edged sword.氧化基因组损伤及其在神经退行性疾病中的修复:过渡金属的双刃剑功能。
J Alzheimers Dis. 2011;24 Suppl 2(0 2):183-98. doi: 10.3233/JAD-2011-110281.
8
Dynamic recruitment of licensing factor Cdt1 to sites of DNA damage.许可因子 Cdt1 动态募集到 DNA 损伤部位。
J Cell Sci. 2011 Feb 1;124(Pt 3):422-34. doi: 10.1242/jcs.074229. Epub 2011 Jan 11.
9
Use of γH2AX and other biomarkers of double-strand breaks during radiotherapy.放射治疗中 γH2AX 和其他双链断裂生物标志物的应用。
Semin Radiat Oncol. 2010 Oct;20(4):223-31. doi: 10.1016/j.semradonc.2010.05.004.
10
DNA damage signaling in response to double-strand breaks during mitosis.有丝分裂过程中双链断裂所诱导的 DNA 损伤信号转导。
J Cell Biol. 2010 Jul 26;190(2):197-207. doi: 10.1083/jcb.200911156.