Demeke Taye, El-Gawad Gamal Abd, Osmancevic Amra, Gillstedt Martin, Landin-Wilhelmsen Kerstin
Hjällbo Primary Health care, S-424 32, Gothenburg, Sweden,
Arch Osteoporos. 2015;10(1):208. doi: 10.1007/s11657-015-0208-5. Epub 2015 Feb 19.
Vitamin D deficiency can lead to osteomalacia. Bone mineral density was lower in Somali women, living in Sweden, in relation to both the American and the African-American reference populations. The majority, 73 %, had vitamin D deficiency, and supplementation should be considered to prevent from osteomalacia, osteoporosis and future fractures.
Low vitamin D can lead to osteomalacia. The hypothesis was that bone mineral density (BMD) in Somali women living in Sweden was lower in comparison with different ethnic reference populations.
Women from Somalia, n = 67, median age 35.8 years (range 18 to 56), latitude 0-10° North living in Gothenburg, Sweden, latitude 57° North, >2 years were studied. All wore traditional Islamic clothing and had skin photo type V. BMD was recorded as the Z-score and compared with white American and African-American women, respectively, using standard data from the dual energy X-ray absorptiometry (DXA) manufacturer (Lunar Prodigy enCORETM, GE Healthcare, LU44663). A fasting blood test was drawn for analysis of serum 25(OH)D.
The median Z-score compared with the American white population was -0.9 SD of the lumbar spine (p < 0.00001), 0.1 SD of the left hip and 0.0 SD of the right hip (ns). The median Z-score compared with the African-American population was -1.6 SD of the lumbar spine (p < 0.00001), -0.9 SD of the left hip and -0.9 SD of the right hip (p < 0.001). The majority, 73 %, had vitamin D deficiency, serum 25(OH)D <25 nmol/l (<10 ng/ml). BMD did not correlate to vitamin D levels or to the number of years in Sweden. One wrist fracture was reported.
BMD was lower in these fairly young immigrant women from Somalia, living in Sweden, in relation to both the American and the African-American reference populations. Vitamin D supplementation should be considered to prevent from osteomalacia, osteoporosis and future fractures.
维生素D缺乏可导致骨软化症。居住在瑞典的索马里女性的骨矿物质密度低于美国和非裔美国参考人群。大多数（73%）存在维生素D缺乏，应考虑补充维生素D以预防骨软化症、骨质疏松症及未来骨折。
低维生素D可导致骨软化症。研究假设是居住在瑞典的索马里女性的骨矿物质密度（BMD）低于不同种族参考人群。
对来自索马里的67名女性进行研究，年龄中位数35.8岁（范围18至56岁），居住在瑞典哥德堡（北纬57°），来自北纬0 - 10°，居住时间超过2年。所有女性均穿着传统伊斯兰服装，皮肤光型为V型。BMD记录为Z值，并分别与美国白人女性和非裔美国女性进行比较，使用双能X线吸收法（DXA）制造商（通用电气医疗集团Lunar Prodigy enCORETM，LU44663）的标准数据。抽取空腹血检测血清25(OH)D。
与美国白人人群相比，腰椎的Z值中位数为 -0.9标准差（p < 0.00001），左髋为0.1标准差，右髋为0.0标准差（无统计学意义）。与非裔美国人群相比，腰椎的Z值中位数为 -１.6标准差（p < 0.00001），左髋为 -0.9标准差，右髋为 -0.9标准差（p < 0.001）。大多数（73%）存在维生素D缺乏，血清25(OH)D < 25 nmol/l（< 10 ng/ml）。BMD与维生素D水平或在瑞典居住的年数无关。报告了1例腕部骨折。
这些居住在瑞典的相当年轻的索马里移民女性相对于美国和非裔美国参考人群，其BMD较低。应考虑补充维生素D以预防骨软化症、骨质疏松症及未来骨折。
