McDonald A J, Pearson J C
Department of Anatomy, University of South Carolina School of Medicine, Columbia 29208.
Neurosci Lett. 1989 May 22;100(1-3):53-8. doi: 10.1016/0304-3940(89)90659-9.
Colocalization of gamma-aminobutyric acid (GABA) immunoreactivity with somatostatin (SOM), neuropeptide Y (NPY), cholecystokinin (CCK), and vasoactive intestinal polypeptide (VIP) immunoreactivity was demonstrated in non-pyramidal neurons of the basolateral amygdala using a two-color immunoperoxidase procedure. Approximately 80-90% of SOM- and NPY-positive neurons in the basolateral amygdala were also immunoreactive for GABA. Virtually all large CCK-positive neurons also exhibited GABA-like immunoreactivity. About one-half of VIP-positive neurons and small CCK-positive cells were also immunoreactive for GABA.
采用双色免疫过氧化物酶法,在基底外侧杏仁核的非锥体神经元中证实了γ-氨基丁酸(GABA)免疫反应性与生长抑素(SOM)、神经肽Y(NPY)、胆囊收缩素(CCK)和血管活性肠肽(VIP)免疫反应性的共定位。基底外侧杏仁核中约80-90%的SOM和NPY阳性神经元也对GABA呈免疫反应性。几乎所有大的CCK阳性神经元也表现出GABA样免疫反应性。约一半的VIP阳性神经元和小的CCK阳性细胞也对GABA呈免疫反应性。