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亨廷顿舞蹈病zQ175小鼠模型中HTT包涵体大小、位置及时间特征:一项体内高内涵成像研究

Characterization of HTT inclusion size, location, and timing in the zQ175 mouse model of Huntington's disease: an in vivo high-content imaging study.

作者信息

Carty Nikisha, Berson Nadège, Tillack Karsten, Thiede Christina, Scholz Diana, Kottig Karsten, Sedaghat Yalda, Gabrysiak Christina, Yohrling George, von der Kammer Heinz, Ebneth Andreas, Mack Volker, Munoz-Sanjuan Ignacio, Kwak Seung

机构信息

Evotec AG, Manfred Eigen Campus, Hamburg, Germany.

CHDI Management/CHDI Foundation, Princeton, New Jersey, United States of America.

出版信息

PLoS One. 2015 Apr 10;10(4):e0123527. doi: 10.1371/journal.pone.0123527. eCollection 2015.

DOI:10.1371/journal.pone.0123527
PMID:25859666
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4393127/
Abstract

Huntington's disease (HD) is an autosomal dominant neurodegenerative disorder caused by a CAG trinucleotide repeat expansion in the huntingtin gene. Major pathological hallmarks of HD include inclusions of mutant huntingtin (mHTT) protein, loss of neurons predominantly in the caudate nucleus, and atrophy of multiple brain regions. However, the early sequence of histological events that manifest in region- and cell-specific manner has not been well characterized. Here we use a high-content histological approach to precisely monitor changes in HTT expression and characterize deposition dynamics of mHTT protein inclusion bodies in the recently characterized zQ175 knock-in mouse line. We carried out an automated multi-parameter quantitative analysis of individual cortical and striatal cells in tissue slices from mice aged 2-12 months and confirmed biochemical reports of an age-associated increase in mHTT inclusions in this model. We also found distinct regional and subregional dynamics for inclusion number, size and distribution with subcellular resolution. We used viral-mediated suppression of total HTT in the striatum of zQ175 mice as an example of a therapeutically-relevant but heterogeneously transducing strategy to demonstrate successful application of this platform to quantitatively assess target engagement and outcome on a cellular basis.

摘要

亨廷顿舞蹈症(HD)是一种常染色体显性神经退行性疾病,由亨廷顿基因中的CAG三核苷酸重复序列扩增引起。HD的主要病理特征包括突变型亨廷顿蛋白(mHTT)的包涵体、主要在尾状核中的神经元丧失以及多个脑区的萎缩。然而,以区域和细胞特异性方式表现的组织学事件的早期序列尚未得到很好的表征。在这里,我们使用高内涵组织学方法来精确监测HTT表达的变化,并表征最近鉴定的zQ175基因敲入小鼠品系中mHTT蛋白包涵体的沉积动态。我们对2至12个月龄小鼠组织切片中的单个皮质和纹状体细胞进行了自动多参数定量分析,并证实了该模型中mHTT包涵体随年龄增长增加的生化报告。我们还发现了包涵体数量、大小和分布在亚细胞分辨率下不同的区域和亚区域动态。我们以病毒介导的zQ175小鼠纹状体中总HTT的抑制为例,这是一种与治疗相关但转导不均一的策略,以证明该平台成功应用于在细胞基础上定量评估靶点结合和结果。

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本文引用的文献

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Characterization of neurophysiological and behavioral changes, MRI brain volumetry and 1H MRS in zQ175 knock-in mouse model of Huntington's disease.亨廷顿病 ZQ175 基因敲入小鼠模型的神经生理和行为变化特征、MRI 脑容量测定和 1H MRS。
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