糖尿病相关痴呆中晚期糖基化终产物的循环水平 - Suppr

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超能文献

Circulating Levels of Advanced Glycation End Products in Diabetes Mellitus-Related Dementia.

作者信息

Hirose Daisuke, Hanyu Haruo, Fukusawa Raita, Hatanaka Hirokuni, Namioka Nayuta, Okita Misa

机构信息

Department of Geriatric Medicine, Tokyo Medical University, Tokyo, Japan.

出版信息

J Am Geriatr Soc. 2015 Oct;63(10):2196-8. doi: 10.1111/jgs.13683.

DOI:10.1111/jgs.13683

PMID:26480987

Abstract

摘要

相似文献

Circulating Levels of Advanced Glycation End Products in Diabetes Mellitus-Related Dementia.糖尿病相关痴呆中晚期糖基化终产物的循环水平

J Am Geriatr Soc. 2015 Oct;63(10):2196-8. doi: 10.1111/jgs.13683.

Association of peripheral neuropathy with circulating advanced glycation end products, soluble receptor for advanced glycation end products and other risk factors in patients with type 2 diabetes.2 型糖尿病患者周围神经病变与循环晚期糖基化终产物、可溶性晚期糖基化终产物受体及其他危险因素的关系。

Diabetes Metab Res Rev. 2014 Nov;30(8):679-85. doi: 10.1002/dmrr.2529.

Serum levels of soluble form of receptor for advanced glycation end products (sRAGE) are positively associated with circulating AGEs and soluble form of VCAM-1 in patients with type 2 diabetes.在2型糖尿病患者中，血清晚期糖基化终末产物受体的可溶性形式（sRAGE）水平与循环中的晚期糖基化终末产物（AGEs）及血管细胞黏附分子-1的可溶性形式呈正相关。

Microvasc Res. 2008 May;76(1):52-6. doi: 10.1016/j.mvr.2007.09.004. Epub 2007 Oct 5.

Atorvastatin decreases serum levels of advanced glycation end products (AGEs) in patients with type 2 diabetes.阿托伐他汀可降低2型糖尿病患者血清晚期糖基化终末产物（AGEs）水平。

Clin Exp Med. 2006 Dec;6(4):191-3. doi: 10.1007/s10238-006-0115-6.

Serum or cerebrospinal fluid levels of glyceraldehyde-derived advanced glycation end products (AGEs) may be a promising biomarker for early detection of Alzheimer's disease.甘油醛衍生的晚期糖基化终产物（AGEs）的血清或脑脊液水平可能是早期检测阿尔茨海默病的一种有前景的生物标志物。

Med Hypotheses. 2005;64(6):1205-7. doi: 10.1016/j.mehy.2005.01.016.

Decreased plasma levels of soluble low density lipoprotein receptor-related protein-1 (sLRP) and the soluble form of the receptor for advanced glycation end products (sRAGE) in the clinical diagnosis of Alzheimer's disease.在阿尔茨海默病的临床诊断中，血浆中可溶性低密度脂蛋白受体相关蛋白-1（sLRP）和晚期糖基化终产物受体可溶性形式（sRAGE）的水平降低。

J Clin Neurosci. 2013 Mar;20(3):357-61. doi: 10.1016/j.jocn.2012.06.005. Epub 2012 Dec 7.

Plasma sRAGE is independently associated with high sensitivity C-reactive protein in type 2 diabetes without coronary artery disease.血浆 sRAGE 与 2 型糖尿病无冠心病患者高敏 C 反应蛋白独立相关。

Diabetes Res Clin Pract. 2010 Mar;87(3):e19-22. doi: 10.1016/j.diabres.2009.12.005. Epub 2009 Dec 23.

Increased serum levels of advanced glycation end products in NIDDM patients with diabetic complications.患有糖尿病并发症的非胰岛素依赖型糖尿病患者血清晚期糖基化终末产物水平升高。

Diabetes Care. 1998 Jun;21(6):1027.

Are advanced glycation end-products specific biomarkers for Alzheimer's disease?晚期糖基化终产物是阿尔茨海默病的特异性生物标志物吗？

Int J Geriatr Psychiatry. 2005 Sep;20(9):896-7. doi: 10.1002/gps.1372.

Increased serum high-mobility group box-1 and cleaved receptor for advanced glycation endproducts levels and decreased endogenous secretory receptor for advanced glycation endproducts levels in diabetic and non-diabetic patients with heart failure.糖尿病和非糖尿病心力衰竭患者血清高迁移率族蛋白 B1 和晚期糖基化终产物受体裂解产物水平升高，内源性晚期糖基化终产物受体水平降低。

Eur J Heart Fail. 2011 Apr;13(4):440-9. doi: 10.1093/eurjhf/hfq231. Epub 2011 Jan 24.

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A Bibenzyl Component Moscatilin Mitigates Glycation-Mediated Damages in an SH-SY5Y Cell Model of Neurodegenerative Diseases through AMPK Activation and RAGE/NF-B Pathway Suppression.一种联苄成分莫斯卡汀通过激活 AMPK 和抑制 RAGE/NF-κB 通路减轻神经退行性疾病 SH-SY5Y 细胞模型中的糖基化损伤。

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Decreased Muscle Strength and Quality in Diabetes-Related Dementia.糖尿病相关性痴呆患者的肌肉力量和质量下降

Dement Geriatr Cogn Dis Extra. 2017 Dec 22;7(3):454-462. doi: 10.1159/000485177. eCollection 2017 Sep-Dec.

Toxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALD.非酒精性脂肪性肝病和酒精性肝病发病/进展病理生理学的毒性衰老（TAGE）理论

Nutrients. 2017 Jun 20;9(6):634. doi: 10.3390/nu9060634.

Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases.血清毒性 AGEs（TAGE）水平可能是一种有前途的新型生物标志物，用于预测生活方式相关疾病的发生/进展。

Diagnostics (Basel). 2016 Jun 7;6(2):23. doi: 10.3390/diagnostics6020023.

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