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Be resistant to apoptosis: a host factor from gingival fibroblasts.

作者信息

Cheng R, Hu T, Bhowmick N A

机构信息

State Key Laboratory of Oral Diseases, Department of Preventive Dentistry, West China Hospital of Stomatology, Sichuan University, Chengdu, China.

Department of Medicine, Cedars-Sinai Medical Center, Los Angeles, CA, USA.

出版信息

Cell Death Dis. 2015 Dec 3;6(12):e2009. doi: 10.1038/cddis.2015.350.

DOI:10.1038/cddis.2015.350
PMID:26633715
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4720885/
Abstract
摘要

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Cell Death Discov. 2015 Nov 9;1:15046. doi: 10.1038/cddiscovery.2015.46. eCollection 2015.
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J Dent Res. 2014 Sep;93(9):834-9. doi: 10.1177/0022034514544300. Epub 2014 Jul 23.
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Immunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host response.
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Oral Health Prev Dent. 2023 Feb 16;21:49-60. doi: 10.3290/j.ohpd.b3904343.
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Porphyromonas gingivalis Outer Membrane Vesicles Promote Apoptosis via msRNA-Regulated DNA Methylation in Periodontitis.牙龈卟啉单胞菌外膜囊泡通过 msRNA 调控的 DNA 甲基化促进牙周炎中的细胞凋亡。
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