微生物产生的乙酸盐：理解肥胖的“缺失环节”？
Microbially Produced Acetate: A "Missing Link" in Understanding Obesity?
机构信息
Departments of Medicine and Microbiology, New York University Langone Medical Center, New York, NY 10016, USA.
Departments of Medicine and Microbiology, New York University Langone Medical Center, New York, NY 10016, USA; Department of Veterans Affairs Medical Center, New York, NY 10010, USA.
出版信息
Cell Metab. 2016 Jul 12;24(1):9-10. doi: 10.1016/j.cmet.2016.06.023.
Numerous studies have connected the gut microbiome with diet-induced obesity; however, mechanistic explanations for the host-microbial interactions are needed. Perry et al. (2016) present studies suggesting that microbially produced acetate (MPA) increases post-prandial insulin release via a sequential and integrated gut, brain, and pancreatic signaling network promoting energy retention.
许多研究将肠道微生物组与饮食诱导的肥胖联系起来；然而，需要对宿主-微生物相互作用的机制进行解释。Perry 等人（2016 年）提出的研究表明，微生物产生的醋酸盐（MPA）通过促进能量保留的肠道、大脑和胰腺信号网络的顺序和综合作用，增加餐后胰岛素的释放。
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