Experimental Medicine Program, University of British Columbia, Vancouver, British Columbia, Canada.
Centre for Hip Health and Mobility, University of British Columbia, Vancouver, British Columbia, Canada.
Br J Sports Med. 2017 Mar;51(5):428-435. doi: 10.1136/bjsports-2016-096040. Epub 2016 Jul 14.
Injury aetiology models that have evolved over the previous two decades highlight a number of factors which contribute to the causal mechanisms for athletic injuries. These models highlight the pathway to injury, including (1) internal risk factors (eg, age, neuromuscular control) which predispose athletes to injury, (2) exposure to external risk factors (eg, playing surface, equipment), and finally (3) an inciting event, wherein biomechanical breakdown and injury occurs. The most recent aetiological model proposed in 2007 was the first to detail the dynamic nature of injury risk, whereby participation may or may not result in injury, and participation itself alters injury risk through adaptation. However, although training and competition workloads are strongly associated with injury, existing aetiology models neither include them nor provide an explanation for how workloads alter injury risk. Therefore, we propose an updated injury aetiology model which includes the effects of workloads. Within this model, internal risk factors are differentiated into modifiable and non-modifiable factors, and workloads contribute to injury in three ways: (1) exposure to external risk factors and potential inciting events, (2) fatigue, or negative physiological effects, and (3) fitness, or positive physiological adaptations. Exposure is determined solely by total load, while positive and negative adaptations are controlled both by total workloads, as well as changes in load (eg, the acute:chronic workload ratio). Finally, we describe how this model explains the load-injury relationships for total workloads, acute:chronic workload ratios and the training load-injury paradox.
在过去二十年中发展起来的损伤病因模型强调了许多因素,这些因素促成了运动损伤的因果机制。这些模型突出了损伤的途径,包括(1)内在风险因素(例如,年龄、神经肌肉控制)使运动员容易受伤,(2)暴露于外在风险因素(例如,场地、设备),最后(3)一个激发事件,其中生物力学的崩溃和损伤发生。2007 年提出的最新病因模型是第一个详细描述损伤风险动态性质的模型,即参与可能导致或不导致损伤,而参与本身通过适应改变损伤风险。然而,尽管训练和比赛工作量与损伤强烈相关,但现有的病因模型既不包括它们,也不解释工作量如何改变损伤风险。因此,我们提出了一个包含工作量影响的更新的损伤病因模型。在这个模型中,内在风险因素分为可改变和不可改变的因素,工作量通过三种方式导致损伤:(1)暴露于外在风险因素和潜在的激发事件,(2)疲劳或负面生理影响,以及(3)健康水平或积极的生理适应。暴露仅由总负荷决定,而积极和消极的适应都由总工作量以及负荷变化(例如,急性:慢性工作量比)控制。最后,我们描述了这个模型如何解释总工作量、急性:慢性工作量比和训练负荷-损伤悖论的负荷-损伤关系。