Modulating Morphine Context-Induced Drug Memory With Deep Brain Stimulation: More Research Questions by Lowering Stimulation Frequencies?
作者信息
Creed Meaghan, Bonci Antonello, Leggio Lorenzo
机构信息
Department of Basic Neurosciences (MC), Medical Faculty, University of Geneva, Geneva, Switzerland.
Synaptic Plasticity Section, National Institute on Drug Abuse Intramural Research Program, Johns Hopkins University School of Medicine, Baltimore; Solomon H. Snyder Neuroscience Institute, Johns Hopkins University School of Medicine, Baltimore; Department of Psychiatry, Johns Hopkins University School of Medicine, Baltimore.
出版信息
Biol Psychiatry. 2016 Nov 1;80(9):647-649. doi: 10.1016/j.biopsych.2016.08.022.
Abstract
摘要
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Deep brain stimulation of the nucleus accumbens shell attenuates cocaine reinstatement through local and antidromic activation.伏隔核壳部的深部脑刺激通过局部和逆行激活来减弱可卡因复吸。
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Rescuing cocaine-induced prefrontal cortex hypoactivity prevents compulsive cocaine seeking.挽救可卡因诱导的前额叶皮层活动低下可预防强迫性可卡因觅药行为。
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Chronic deep brain stimulation in the rat nucleus accumbens and its effect on morphine reinforcement.大鼠伏隔核慢性深部脑刺激及其对吗啡强化作用的影响。
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Repeated electrical stimulation of reward-related brain regions affects cocaine but not "natural" reinforcement.对与奖赏相关的脑区进行反复电刺激会影响可卡因成瘾，但不会影响“自然”强化。
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