• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Susceptibility to chronic social stress increases plaque progression, vulnerability and platelet activation.对慢性社会压力的易感性会增加斑块进展、易损性和血小板活化。
Thromb Haemost. 2017 Apr 3;117(4):816-818. doi: 10.1160/TH16-10-0817. Epub 2017 Jan 12.
2
Hyperreactivity of junctional adhesion molecule A-deficient platelets accelerates atherosclerosis in hyperlipidemic mice.Junctional adhesion molecule A 缺陷型血小板的高反应性加速了高脂血症小鼠的动脉粥样硬化。
Circ Res. 2015 Feb 13;116(4):587-99. doi: 10.1161/CIRCRESAHA.116.304035. Epub 2014 Dec 3.
3
6-Gingerol Ameliorates Behavioral Changes and Atherosclerotic Lesions in ApoE Mice Exposed to Chronic Mild Stress.6-姜酚可改善慢性轻度应激暴露的载脂蛋白 E 小鼠的行为改变和动脉粥样硬化病变。
Cardiovasc Toxicol. 2018 Oct;18(5):420-430. doi: 10.1007/s12012-018-9452-4.
4
Social disruption stress increases IL-6 levels and accelerates atherosclerosis in ApoE-/- mice.社交中断应激增加载脂蛋白 E 基因敲除小鼠的白细胞介素 6 水平并加速动脉粥样硬化。
Atherosclerosis. 2012 Apr;221(2):359-65. doi: 10.1016/j.atherosclerosis.2011.11.041. Epub 2011 Dec 26.
5
Endothelial Cell-Derived Von Willebrand Factor, But Not Platelet-Derived, Promotes Atherosclerosis in Apolipoprotein E-Deficient Mice.内皮细胞衍生的血管性血友病因子而非血小板衍生的促进载脂蛋白 E 缺陷小鼠的动脉粥样硬化。
Arterioscler Thromb Vasc Biol. 2018 Mar;38(3):520-528. doi: 10.1161/ATVBAHA.117.309918. Epub 2018 Jan 18.
6
Exenatide mitigated diet-induced vascular aging and atherosclerotic plaque growth in ApoE-deficient mice under chronic stress.艾塞那肽缓解慢性应激下载脂蛋白 E 缺陷小鼠饮食诱导的血管衰老和动脉粥样硬化斑块生长。
Atherosclerosis. 2017 Sep;264:1-10. doi: 10.1016/j.atherosclerosis.2017.07.014. Epub 2017 Jul 13.
7
Hematopoietic α7 nicotinic acetylcholine receptor deficiency increases inflammation and platelet activation status, but does not aggravate atherosclerosis.造血α7 型烟碱型乙酰胆碱受体缺失增加炎症和血小板激活状态，但不会加重动脉粥样硬化。
J Thromb Haemost. 2015 Jan;13(1):126-35. doi: 10.1111/jth.12765. Epub 2014 Nov 13.
8
Effect of atorvastatin on atherosclerotic plaque formation and platelet activation in hypercholesterolemic rats.阿托伐他汀对高胆固醇血症大鼠动脉粥样硬化斑块形成和血小板活化的影响。
Can J Physiol Pharmacol. 2013 Sep;91(9):680-5. doi: 10.1139/cjpp-2012-0325. Epub 2013 Apr 8.
9
Chronic intermittent mental stress promotes atherosclerotic plaque vulnerability, myocardial infarction and sudden death in mice.慢性间歇性精神应激会增加小鼠动脉粥样硬化斑块的易损性、心肌梗死风险及猝死几率。
Atherosclerosis. 2015 Sep;242(1):288-94. doi: 10.1016/j.atherosclerosis.2015.07.025. Epub 2015 Jul 15.
10
Orthopedic surgery increases atherosclerotic lesions and necrotic core area in ApoE-/- mice.骨科手术会增加载脂蛋白E基因敲除（ApoE-/-）小鼠的动脉粥样硬化病变和坏死核心区域。
Atherosclerosis. 2016 Dec;255:164-170. doi: 10.1016/j.atherosclerosis.2016.07.909. Epub 2016 Oct 28.

引用本文的文献

1
Stress drives myelopoiesis to impair atherosclerosis resolution.应激促使骨髓生成，从而损害动脉粥样硬化的消退。
Res Sq. 2025 Jun 9:rs.3.rs-6727599. doi: 10.21203/rs.3.rs-6727599/v1.
2
Tetraspanin CD37 regulates platelet hyperreactivity and thrombosis.四跨膜蛋白CD37调节血小板高反应性和血栓形成。
Cardiovasc Res. 2025 Jun 12;121(6):943-956. doi: 10.1093/cvr/cvaf051.
3
Convergence of endothelial dysfunction, inflammation and glucocorticoid resistance in depression-related cardiovascular diseases.在与抑郁相关的心血管疾病中，内皮功能障碍、炎症和糖皮质激素抵抗的汇聚。
BMC Immunol. 2024 Sep 27;25(1):61. doi: 10.1186/s12865-024-00653-9.
4
Chronic social defeat alters brain vascular-associated cell gene expression patterns leading to vascular dysfunction and immune system activation.慢性社会挫败会改变与大脑血管相关的细胞基因表达模式，导致血管功能障碍和免疫系统激活。
J Neuroinflammation. 2023 Jun 28;20(1):154. doi: 10.1186/s12974-023-02827-5.
5
Evidence and Characteristics of Traditional Chinese Medicine for Coronary Heart Disease Patients With Anxiety or Depression: A Meta-Analysis and Systematic Review.冠心病合并焦虑或抑郁患者的中医证据及特征：一项Meta分析与系统评价
Front Pharmacol. 2022 May 5;13:854292. doi: 10.3389/fphar.2022.854292. eCollection 2022.
6
Recent Progress of Chronic Stress in the Development of Atherosclerosis.慢性应激在动脉粥样硬化发生发展中的最新进展
Oxid Med Cell Longev. 2022 Mar 8;2022:4121173. doi: 10.1155/2022/4121173. eCollection 2022.
7
Chronic Stress A Potential Suspect Zero of Atherosclerosis: A Systematic Review.慢性应激——动脉粥样硬化的一个潜在可疑病因：一项系统评价
Front Cardiovasc Med. 2021 Dec 20;8:738654. doi: 10.3389/fcvm.2021.738654. eCollection 2021.
8
Analysis of cerebrovascular dysfunction caused by chronic social defeat in mice.分析慢性社交挫败导致的小鼠脑血管功能障碍。
Brain Behav Immun. 2020 Aug;88:735-747. doi: 10.1016/j.bbi.2020.05.030. Epub 2020 May 12.
9
Inflaming sex differences in mood disorders.情绪障碍中炎症导致的性别差异。
Neuropsychopharmacology. 2019 Jan;44(1):184-199. doi: 10.1038/s41386-018-0124-7. Epub 2018 Jun 19.
10
Apocynin Prevents Abnormal Megakaryopoiesis and Platelet Activation Induced by Chronic Stress.阿朴酯素可预防慢性应激引起的异常巨核细胞生成和血小板活化。
Oxid Med Cell Longev. 2017;2017:9258937. doi: 10.1155/2017/9258937. Epub 2017 Nov 28.

本文引用的文献

1
Chronic intermittent mental stress promotes atherosclerotic plaque vulnerability, myocardial infarction and sudden death in mice.慢性间歇性精神应激会增加小鼠动脉粥样硬化斑块的易损性、心肌梗死风险及猝死几率。
Atherosclerosis. 2015 Sep;242(1):288-94. doi: 10.1016/j.atherosclerosis.2015.07.025. Epub 2015 Jul 15.
2
Individual differences in the peripheral immune system promote resilience versus susceptibility to social stress.外周免疫系统的个体差异促进了对社会压力的恢复力和易感性。
Proc Natl Acad Sci U S A. 2014 Nov 11;111(45):16136-41. doi: 10.1073/pnas.1415191111. Epub 2014 Oct 20.
3
Contribution of homeostatic chemokines CCL19 and CCL21 and their receptor CCR7 to coronary artery disease.稳态趋化因子 CCL19 和 CCL21 及其受体 CCR7 对冠状动脉疾病的贡献。
Arterioscler Thromb Vasc Biol. 2014 Sep;34(9):1933-41. doi: 10.1161/ATVBAHA.113.303081. Epub 2014 Jul 2.
4
Chronic variable stress activates hematopoietic stem cells.慢性可变应激激活造血干细胞。
Nat Med. 2014 Jul;20(7):754-758. doi: 10.1038/nm.3589. Epub 2014 Jun 22.
5
A new murine model of stress-induced complex atherosclerotic lesions.一种应激诱导的复杂动脉粥样硬化病变的新鼠模型。
Dis Model Mech. 2013 Mar;6(2):323-31. doi: 10.1242/dmm.009977. Epub 2013 Jan 11.
6
The worldwide environment of cardiovascular disease: prevalence, diagnosis, therapy, and policy issues: a report from the American College of Cardiology.心血管疾病的全球环境：流行情况、诊断、治疗和政策问题：美国心脏病学会的报告。
J Am Coll Cardiol. 2012 Dec 25;60(25 Suppl):S1-49. doi: 10.1016/j.jacc.2012.11.002.
7
Increased levels of the homeostatic chemokine CXCL13 in human atherosclerosis - Potential role in plaque stabilization.趋化因子 CXCL13 在人类动脉粥样硬化中的水平升高 - 在斑块稳定中的潜在作用。
Atherosclerosis. 2012 Sep;224(1):266-73. doi: 10.1016/j.atherosclerosis.2012.06.071. Epub 2012 Jul 20.
8
Effect of Hurricane Katrina on incidence of acute myocardial infarction in New Orleans three years after the storm.卡特里娜飓风对风暴后三年新奥尔良急性心肌梗死发病率的影响。
Am J Cardiol. 2012 Feb 15;109(4):502-5. doi: 10.1016/j.amjcard.2011.09.045. Epub 2011 Dec 6.
9
A standardized protocol for repeated social defeat stress in mice.一种用于小鼠重复社交挫败应激的标准化方案。
Nat Protoc. 2011 Jul 21;6(8):1183-91. doi: 10.1038/nprot.2011.361.
10
Genetic deletion of chemokine receptor Ccr6 decreases atherogenesis in ApoE-deficient mice.基因敲除趋化因子受体 Ccr6 可减少载脂蛋白 E 缺陷小鼠的动脉粥样硬化形成。
Circ Res. 2011 Aug 5;109(4):374-81. doi: 10.1161/CIRCRESAHA.111.242578. Epub 2011 Jun 16.

Susceptibility to chronic social stress increases plaque progression, vulnerability and platelet activation.

作者信息

Giannarelli Chiara, Rodriguez David T, Zafar M Urooj, Christoffel Daniel, Vialou Vincent, Peña Catherine, Badimon Ana, Hodes Georgia F, Mury Pauline, Rabkin Jacklyn, Alique Matilde, Villa Giulia, Argmann Carmen, Nestler Eric J, Russo Scott J, Badimon Juan J

机构信息

Chiara Giannarelli, MD, PhD, Cardiovascular Research Center, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY 10029, USA, Tel.: +1 212 241 4984, Fax: +1 212 426 6962, E-mail:

Juan J. Badimon, PhD, AtheroThrombosis Research Unit, Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY 10029, USA, Tel.: +1 212 241 8484, Fax: +1 212 426 6962, E-mail:

出版信息

Thromb Haemost. 2017 Apr 3;117(4):816-818. doi: 10.1160/TH16-10-0817. Epub 2017 Jan 12.

DOI:10.1160/TH16-10-0817
PMID:28078352
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5490439/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/dbc0/5490439/ccc91effad1f/nihms865889f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/dbc0/5490439/ccc91effad1f/nihms865889f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/dbc0/5490439/ccc91effad1f/nihms865889f1.jpg