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本文引用的文献

1
Convergent ERK1/2, p38 and JNK mitogen activated protein kinases (MAPKs) signalling mediate catecholoestradiol-induced proliferation of ovine uterine artery endothelial cells.趋同的细胞外信号调节激酶1/2（ERK1/2）、p38和应激活化蛋白激酶（JNK）丝裂原活化蛋白激酶（MAPK）信号传导介导儿茶酚雌二醇诱导的绵羊子宫动脉内皮细胞增殖。
J Physiol. 2017 Jul 15;595(14):4663-4676. doi: 10.1113/JP274119. Epub 2017 Jun 5.
2
Aberrant synthesis, metabolism, and plasma accumulation of circulating estrogens and estrogen metabolites in preeclampsia implications for vascular dysfunction.子痫前期患者循环雌激素及其代谢物的合成、代谢和血浆蓄积异常与血管功能障碍的关系。
Hypertension. 2013 Feb;61(2):480-7. doi: 10.1161/HYPERTENSIONAHA.111.201624. Epub 2013 Jan 14.
3
A novel role for an endothelial adrenergic receptor system in mediating catecholestradiol-induced proliferation of uterine artery endothelial cells.内皮肾上腺素能受体系统在介导儿茶酚雌二醇诱导的子宫动脉内皮细胞增殖中的新作用。
Hypertension. 2011 Nov;58(5):874-81. doi: 10.1161/HYPERTENSIONAHA.111.178046. Epub 2011 Sep 26.
4
Estradiol-17beta and its cytochrome P450- and catechol-O-methyltransferase-derived metabolites stimulate proliferation in uterine artery endothelial cells: role of estrogen receptor-alpha versus estrogen receptor-beta.雌二醇-17β及其细胞色素 P450 和儿茶酚-O-甲基转移酶衍生代谢物刺激子宫动脉内皮细胞增殖：雌激素受体-α与雌激素受体-β的作用。
Hypertension. 2010 Apr;55(4):1005-11. doi: 10.1161/HYPERTENSIONAHA.109.146399. Epub 2010 Mar 8.
5
Deficiency in catechol-O-methyltransferase and 2-methoxyoestradiol is associated with pre-eclampsia.儿茶酚-O-甲基转移酶和2-甲氧基雌二醇缺乏与先兆子痫有关。
Nature. 2008 Jun 19;453(7198):1117-21. doi: 10.1038/nature06951. Epub 2008 May 11.

Angiogenesis during pregnancy: all routes lead to MAPKs.

作者信息

Hu Xiang-Qun, Zhang Lubo

机构信息

Lawrence D. Longo, MD Center for Perinatal Biology, Department of Basic Sciences, Loma Linda University School of Medicine, Loma Linda, CA, USA.

出版信息

J Physiol. 2017 Jul 15;595(14):4571-4572. doi: 10.1113/JP274489. Epub 2017 Jun 8.

DOI:10.1113/JP274489
PMID:28513857
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5509881/
Abstract
摘要