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本文引用的文献

1
Smoking-Dependent Distal-to-Proximal Repatterning of the Adult Human Small Airway Epithelium.吸烟导致的成人人小气道上皮从远端到近端的重塑
Am J Respir Crit Care Med. 2017 Aug 1;196(3):340-352. doi: 10.1164/rccm.201608-1672OC.
2
EGF-Amphiregulin Interplay in Airway Stem/Progenitor Cells Links the Pathogenesis of Smoking-Induced Lesions in the Human Airway Epithelium.气道干/祖细胞中表皮生长因子-双调蛋白的相互作用与人类气道上皮吸烟诱导性病变的发病机制相关。
Stem Cells. 2017 Mar;35(3):824-837. doi: 10.1002/stem.2512. Epub 2016 Nov 17.
3
Rhinovirus Delays Cell Repolarization in a Model of Injured/Regenerating Human Airway Epithelium.鼻病毒在人呼吸道上皮损伤/再生模型中延迟细胞复极化。
Am J Respir Cell Mol Biol. 2016 Oct;55(4):487-499. doi: 10.1165/rcmb.2015-0243OC.
4
Imprinting of the COPD airway epithelium for dedifferentiation and mesenchymal transition.COPD 气道上皮的印记诱导去分化和间充质转化。
Eur Respir J. 2015 May;45(5):1258-72. doi: 10.1183/09031936.00135814. Epub 2015 Mar 5.
5
Airway Basal stem/progenitor cells have diminished capacity to regenerate airway epithelium in chronic obstructive pulmonary disease.气道基底干细胞/祖细胞在慢性阻塞性肺疾病中再生气道上皮的能力减弱。
Am J Respir Crit Care Med. 2014 Oct 15;190(8):955-8. doi: 10.1164/rccm.201406-1167LE.
6
A randomized controlled trial of inhaled corticosteroids (ICS) on markers of epithelial-mesenchymal transition (EMT) in large airway samples in COPD: an exploratory proof of concept study.吸入性糖皮质激素（ICS）对慢性阻塞性肺疾病（COPD）大气道样本中上皮-间质转化（EMT）标志物影响的随机对照试验：一项探索性概念验证研究。
Int J Chron Obstruct Pulmon Dis. 2014 May 27;9:533-42. doi: 10.2147/COPD.S63911. eCollection 2014.
7
Abnormalities in airway epithelial junction formation in chronic obstructive pulmonary disease.慢性阻塞性肺疾病中气道上皮连接形成异常。
Am J Respir Crit Care Med. 2014 Jun 1;189(11):1439-42. doi: 10.1164/rccm.201311-1982LE.
8
EGF shifts human airway basal cell fate toward a smoking-associated airway epithelial phenotype.表皮生长因子使人类气道基底细胞向与吸烟相关的气道上皮表型转变。
Proc Natl Acad Sci U S A. 2013 Jul 16;110(29):12102-7. doi: 10.1073/pnas.1303058110. Epub 2013 Jul 1.
9
The role of uPAR in epithelial-mesenchymal transition in small airway epithelium of patients with chronic obstructive pulmonary disease.尿激酶型纤溶酶原激活物受体在慢性阻塞性肺疾病患者小气道上皮细胞上皮-间充质转化中的作用。
Respir Res. 2013 Jun 28;14(1):67. doi: 10.1186/1465-9921-14-67.
10
Epithelial to mesenchymal transition is increased in patients with COPD and induced by cigarette smoke.COPD 患者的上皮间质转化增加，并且由香烟烟雾诱导。
Thorax. 2013 May;68(5):410-20. doi: 10.1136/thoraxjnl-2012-201761. Epub 2013 Jan 7.

Reply: Epithelial-Mesenchymal Transition: A Necessary New Therapeutic Target in Chronic Obstructive Pulmonary Disease?

作者信息

Yang Jing, Shaykhiev Renat

机构信息

1 West China Hospital of Sichuan University Chengdu, China and.

2 Weill Cornell Medical College New York, New York.

出版信息

Am J Respir Crit Care Med. 2017 Aug 1;196(3):394-395. doi: 10.1164/rccm.201705-0981LE.

DOI:10.1164/rccm.201705-0981LE
PMID:28640654
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5549873/
Abstract
摘要