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From tubular sublimate nephropathy via urinary concentrating mechanism to glomerular disease-Wilhelm Kriz's contribution to modern nephrology : On the occasion of a symposium honoring Wilhelm Kriz on his 80th birthday.

作者信息

Bachmann Sebastian

机构信息

Department of Anatomy, Charité Universitätsmedizin, 10115, Berlin, Germany.

Institut für Vegetative Anatomie, Charité - Universitätsmedizin Berlin, Campus Charité-Mitte, Philippstr. 12, 10115, Berlin, Germany.

出版信息

Pflugers Arch. 2017 Aug;469(7-8):847-857. doi: 10.1007/s00424-017-2010-2. Epub 2017 Jun 24.

DOI:10.1007/s00424-017-2010-2
PMID:28647852
Abstract
摘要

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本文引用的文献

1
Potential relevance of shear stress for slit diaphragm and podocyte function.切应力对裂孔隔膜和足细胞功能的潜在相关性。
Kidney Int. 2017 Jun;91(6):1283-1286. doi: 10.1016/j.kint.2017.02.032.
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New Insights into Podocyte Biology in Glomerular Health and Disease.肾小球健康与疾病中足细胞生物学的新见解
J Am Soc Nephrol. 2017 Jun;28(6):1707-1715. doi: 10.1681/ASN.2017010027. Epub 2017 Apr 12.
3
Accumulation of worn-out GBM material substantially contributes to mesangial matrix expansion in diabetic nephropathy.衰老的肾小球基底膜（GBM）物质的积累在很大程度上导致了糖尿病肾病中系膜基质的扩张。
Am J Physiol Renal Physiol. 2017 Jun 1;312(6):F1101-F1111. doi: 10.1152/ajprenal.00020.2017. Epub 2017 Feb 22.
4
Mechanical challenges to the glomerular filtration barrier: adaptations and pathway to sclerosis.肾小球滤过屏障的机械性挑战：适应与硬化途径
Pediatr Nephrol. 2017 Mar;32(3):405-417. doi: 10.1007/s00467-016-3358-9. Epub 2016 Mar 23.
5
Structural analysis of how podocytes detach from the glomerular basement membrane under hypertrophic stress.足细胞在肥大应激下如何从肾小球基底膜脱离的结构分析。
Front Endocrinol (Lausanne). 2014 Dec 12;5:207. doi: 10.3389/fendo.2014.00207. eCollection 2014.
6
A potential role for mechanical forces in the detachment of podocytes and the progression of CKD.机械力在足细胞脱离及慢性肾脏病进展中的潜在作用。
J Am Soc Nephrol. 2015 Feb;26(2):258-69. doi: 10.1681/ASN.2014030278. Epub 2014 Jul 24.
7
Renal epithelial injury and fibrosis.肾上皮损伤与纤维化。
Biochim Biophys Acta. 2013 Jul;1832(7):931-9. doi: 10.1016/j.bbadis.2013.02.010. Epub 2013 Mar 1.
8
The podocyte's response to stress: the enigma of foot process effacement.足细胞对压力的反应：足突融合之谜。
Am J Physiol Renal Physiol. 2013 Feb 15;304(4):F333-47. doi: 10.1152/ajprenal.00478.2012. Epub 2012 Dec 12.
9
Epithelial-mesenchymal transition (EMT) in kidney fibrosis: fact or fantasy?肾脏纤维化中的上皮-间充质转化（EMT）：事实还是幻想？
J Clin Invest. 2011 Feb;121(2):468-74. doi: 10.1172/jci44595.
10
Tubular overexpression of transforming growth factor-beta1 induces autophagy and fibrosis but not mesenchymal transition of renal epithelial cells.转化生长因子-β1 的管状过表达诱导肾上皮细胞自噬和纤维化，但不诱导间充质转化。
Am J Pathol. 2010 Aug;177(2):632-43. doi: 10.2353/ajpath.2010.091012. Epub 2010 Jul 8.