耐多药结核病——生物机制、流行病学及管理策略
MULTIDRUG RESISTANT TUBERCULOSIS - BIOMECHANISM, EPIDEMIOLOGY AND MANAGEMENT STRATEGIES.
作者信息
Tewari S C, Kalra S P, Dangwal S, Chatterji R S
机构信息
Senior Adviser, Professor and Head, Dept of Resp Medicine, Military Hospital (CTC), Pune.
Professor and Head, Dept of Medicine, Armed Forces Medical College, Pune 411040.
出版信息
Med J Armed Forces India. 1997 Jul;53(3):207-213. doi: 10.1016/S0377-1237(17)30717-7. Epub 2017 Jun 26.
Abstract
Muitidrug resistant tuberculosis has shown an alarming increase and this assumes added importance in view of the increasing number of HIV infected patients. This article reviews the biomechanism of resistance and discusses the present stategies that are available and recommended to tackle the rising incidence of tuberculosis due to resistant mycobacteria.
摘要
耐多药结核病呈惊人增长态势，鉴于感染艾滋病毒患者数量不断增加，这一情况显得尤为重要。本文综述了耐药的生物机制，并讨论了现有及推荐的应对因耐药分枝杆菌导致结核病发病率上升的当前策略。
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MULTIDRUG RESISTANT TUBERCULOSIS - BIOMECHANISM, EPIDEMIOLOGY AND MANAGEMENT STRATEGIES.耐多药结核病——生物机制、流行病学及管理策略
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引用本文的文献
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inhA, a gene encoding a target for isoniazid and ethionamide in Mycobacterium tuberculosis.inhA，一种编码结核分枝杆菌中异烟肼和乙硫异烟胺靶点的基因。
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Implications of multidrug resistance for the future of short-course chemotherapy of tuberculosis: a molecular study.多重耐药对结核病短程化疗未来的影响：一项分子研究。
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