Dorigo Bruno, Bartoli Vittorio, Grisillo Dario, Beconi Daniela
Istituto di Patologia Medica Base, University of Florence, 50134 Florence, Italy.
Pain. 1979 Apr;6(2):183-190. doi: 10.1016/0304-3959(79)90125-8.
The blocking of trigger points in the calf by the local injection of an anesthetic agent was performed in 15 patients with intermittent claudication. Reactive and exercise hyperemia, work load and duration of exercise were recorded before and after infiltration of the trigger points. Reactive hyperemia does not change, but the exercise tolerance of the leg significantly improves after local blocking of the trigger areas and the exercise hyperemia increases because of the higher work load. The pain pattern of intermittent claudication can be activated by the summation of abnormal input from muscles, due to contraction in the presence of anoxia, with activity from trigger points. Local infiltration of trigger areas blocks theirir activity. The vicious cycle of pain is interrupted and the exercise tolerance of the leg is increased, without improving blood circulation.
对15例间歇性跛行患者进行了局部注射麻醉剂封闭小腿触发点的操作。在触发点浸润前后记录反应性充血、运动性充血、工作量和运动持续时间。反应性充血无变化,但触发区域局部封闭后腿部运动耐量显著提高,且由于工作量增加运动性充血增多。间歇性跛行的疼痛模式可因缺氧时肌肉收缩产生的异常输入与触发点的活动相加而被激活。触发区域的局部浸润可阻断其活动。疼痛的恶性循环被打断,腿部运动耐量增加,而血液循环并未改善。