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The status of late-onset vacuous chewing/perioral movements during long-term neuroleptic treatment in rodents: tardive dyskinesia or dystonia?

作者信息

Waddington J L, Molloy A G

出版信息

Psychopharmacology (Berl). 1987;91(1):136-7. doi: 10.1007/BF00690944.

DOI:10.1007/BF00690944
PMID:2881317
Abstract
摘要

相似文献

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The status of late-onset vacuous chewing/perioral movements during long-term neuroleptic treatment in rodents: tardive dyskinesia or dystonia?啮齿动物长期使用抗精神病药物治疗期间迟发性空嚼/口周运动的状况:迟发性运动障碍还是肌张力障碍?
Psychopharmacology (Berl). 1987;91(1):136-7. doi: 10.1007/BF00690944.
2
Effect of 5-HT1A and 5-HT2A/2C receptor modulation on neuroleptic-induced vacuous chewing movements.5-HT1A和5-HT2A/2C受体调节对抗精神病药物诱导的空嚼运动的影响。
Eur J Pharmacol. 2001 Sep 28;428(1):81-6. doi: 10.1016/s0014-2999(01)01284-5.
3
Tardive dystonia and severe tardive dyskinesia. A comparison of risk factors and prognosis.迟发性肌张力障碍和严重迟发性运动障碍。危险因素及预后的比较。
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Pharmacological and neurochemical differences between acute and tardive vacuous chewing movements induced by haloperidol.氟哌啶醇所致急性和迟发性空口咀嚼运动的药理学及神经化学差异
Psychopharmacology (Berl). 1996 Oct;127(4):337-45. doi: 10.1007/s002130050095.
5
[Tardive dystonia: a rare side effect after long-term neuroleptic treatment].
Nervenarzt. 1982 Nov;53(11):674-6.
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Animal models of tardive dyskinesia--a review.迟发性运动障碍的动物模型——综述
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Parallels between behavioral and neurochemical variability in the rat vacuous chewing movement model of tardive dyskinesia.迟发性运动障碍大鼠空嚼运动模型中行为和神经化学变异性的比较。
Behav Brain Res. 2012 Jun 1;231(2):323-36. doi: 10.1016/j.bbr.2012.03.045. Epub 2012 Apr 6.
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引用本文的文献

1
Electromyographical differentiation of the components of perioral movements induced by SKF 38393 and physostigmine in the rat.大鼠中由SKF 38393和毒扁豆碱诱导的口周运动成分的肌电图分化
Psychopharmacology (Berl). 1993;112(4):428-36. doi: 10.1007/BF02244890.
2
Vacuous jaw movements induced by sub-chronic administration of haloperidol: interactions with scopolamine.氟哌啶醇亚慢性给药诱导的无意义下颌运动:与东莨菪碱的相互作用
Psychopharmacology (Berl). 1993;111(1):99-105. doi: 10.1007/BF02257414.
3
Characteristics of oral movements in rats during and after chronic haloperidol and fluphenazine administration.

本文引用的文献

1
Dopamine receptor function and spontaneous orofacial dyskinesia in rats during 6-month neuroleptic treatments.6个月抗精神病药物治疗期间大鼠的多巴胺受体功能与自发性口面部运动障碍
Adv Biochem Psychopharmacol. 1983;37:299-308.
2
Spontaneous orofacial dyskinesia and dopaminergic function in rats after 6 months of neuroleptic treatment.抗精神病药物治疗6个月后大鼠的自发性口面部运动障碍与多巴胺能功能
Science. 1983 Apr 29;220(4596):530-2. doi: 10.1126/science.6132447.
3
Acute dystonia induced by neuroleptic drugs.抗精神病药物所致急性肌张力障碍
慢性给予氟哌啶醇和氟奋乃静期间及之后大鼠口腔运动的特征
Psychopharmacology (Berl). 1988;94(3):421-7. doi: 10.1007/BF00174701.
4
Interaction between chronic amphetamine and neuroleptic treatments on oral behavior in rats.慢性苯丙胺与抗精神病药物联合治疗对大鼠口腔行为的影响
Psychopharmacology (Berl). 1987;93(2):218-22. doi: 10.1007/BF00179937.
5
The ageing brain, neuroleptic drugs and the enigma of schizophrenia.衰老的大脑、抗精神病药物与精神分裂症之谜
Ir J Med Sci. 1988 May;157(5):135-41. doi: 10.1007/BF02949282.
6
Neuroleptic-induced oral dyskinesias: effects of progabide and lack of correlation with regional changes in glutamic acid decarboxylase and choline acetyltransferase activities.抗精神病药物所致口腔运动障碍:普罗加比的作用及与谷氨酸脱羧酶和胆碱乙酰转移酶活性区域变化的无关性
Psychopharmacology (Berl). 1987;93(1):94-100. doi: 10.1007/BF02439593.
7
Chronic neuroleptic-induced mouth movements in the rat: suppression by CCK and selective dopamine D1 and D2 receptor antagonists.大鼠慢性抗精神病药物诱导的口腔运动:胆囊收缩素及选择性多巴胺D1和D2受体拮抗剂的抑制作用
Psychopharmacology (Berl). 1989;98(3):372-9. doi: 10.1007/BF00451690.
8
Intermittent and continuous haloperidol regimens produce different types of oral dyskinesias in rats.间歇性和连续性氟哌啶醇给药方案在大鼠中会产生不同类型的口腔运动障碍。
Psychopharmacology (Berl). 1990;100(3):404-12. doi: 10.1007/BF02244615.
9
Drug-induced purposeless chewing: animal model of dyskinesia or nausea?
Psychopharmacology (Berl). 1990;102(3):325-8. doi: 10.1007/BF02244098.
10
Spontaneous orofacial movements induced in rodents by very long-term neuroleptic drug administration: phenomenology, pathophysiology and putative relationship to tardive dyskinesia.长期给予抗精神病药物诱导啮齿动物出现的自发性口面部运动:现象学、病理生理学及与迟发性运动障碍的假定关系
Psychopharmacology (Berl). 1990;101(4):431-47. doi: 10.1007/BF02244220.
Psychopharmacology (Berl). 1986;88(4):403-19. doi: 10.1007/BF00178501.
4
Duration of treatment relationships for involuntary movements (tardive dyskinesia): concordance between cross-sectional, clinical, and longitudinal animal studies?非自主运动(迟发性运动障碍)治疗关系的持续时间:横断面研究、临床研究与纵向动物研究之间的一致性?
Arch Gen Psychiatry. 1986 Feb;43(2):191-2. doi: 10.1001/archpsyc.1986.01800020101018.
5
Association of intellectual impairment, negative symptoms, and aging with tardive dyskinesia: clinical and animal studies.智力障碍、阴性症状及衰老与迟发性运动障碍的关联:临床与动物研究
J Clin Psychiatry. 1985 Apr;46(4 Pt 2):29-33.
6
Tardive dyskinesia in the aged. Duration of treatment relationships.老年人迟发性运动障碍。治疗持续时间的关系。
Arch Gen Psychiatry. 1985 Mar;42(3):278-84. doi: 10.1001/archpsyc.1985.01790260072009.