大鼠海马切片中对单脉冲刺激的突触反应的 N-甲基-D-天冬氨酸受体介导成分。 - Suppr

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超能文献

N-methyl-D-aspartate receptor-mediated component of synaptic responses to single-pulse stimulation in rat hippocampal slices.

作者信息

Muller D, Lynch G

机构信息

Center for the Neurobiology of Learning and Memory, University of California, Irvine 92717.

出版信息

Synapse. 1988;2(6):666-8. doi: 10.1002/syn.890020614.

DOI:10.1002/syn.890020614

PMID:2905540

Abstract

摘要

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N-methyl-D-aspartate receptor-mediated component of synaptic responses to single-pulse stimulation in rat hippocampal slices.大鼠海马切片中对单脉冲刺激的突触反应的 N-甲基-D-天冬氨酸受体介导成分。

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Synaptic modulation of N-methyl-D-aspartate receptor mediated responses in hippocampus.海马体中N-甲基-D-天冬氨酸受体介导反应的突触调节

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APV, an N-methyl-D-aspartate receptor antagonist, blocks the hippocampal theta rhythm in behaving rats.APV是一种N-甲基-D-天冬氨酸受体拮抗剂，可阻断行为学实验大鼠的海马θ节律。

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N-methyl-D-aspartate receptor activation is required for the induction of both early and late phases of long-term potentiation in rat hippocampal slices.N-甲基-D-天冬氨酸受体激活是大鼠海马切片中长时程增强早期和晚期诱导所必需的。

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N-methyl-D-aspartate receptor-dependent long-term potentiation in CA1 region affects synaptic expression of glutamate receptor subunits and associated proteins in the whole hippocampus.CA1区中N-甲基-D-天冬氨酸受体依赖性长时程增强作用影响整个海马体中谷氨酸受体亚基及相关蛋白的突触表达。

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Quantitative correlation between tetanus-induced decreases in extracellular calcium and LTP.破伤风诱导的细胞外钙减少与长时程增强之间的定量相关性。

Brain Res. 1988 Jun 28;454(1-2):1-10. doi: 10.1016/0006-8993(88)90798-6.

Evidence for the activation of the N-methyl-D-aspartate receptor during epileptiform discharge.癫痫样放电期间N-甲基-D-天冬氨酸受体激活的证据。

Adv Exp Med Biol. 1986;203:465-74. doi: 10.1007/978-1-4684-7971-3_35.

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The 1980s: D-AP5, LTP and a Decade of NMDA Receptor Discoveries.20 世纪 80 年代：D-AP5、LTP 和十年的 NMDA 受体发现。

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Blockade of long-term potentiation and of NMDA receptors by the protein kinase C antagonist calphostin C.

Naunyn Schmiedebergs Arch Pharmacol. 1993 Jul;348(1):1-6. doi: 10.1007/BF00168529.

Protein kinase C activity is not responsible for the expression of long-term potentiation in hippocampus.蛋白激酶C的活性与海马体中长时程增强效应的表达无关。

Proc Natl Acad Sci U S A. 1990 Jun;87(11):4073-7. doi: 10.1073/pnas.87.11.4073.

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