伯基特淋巴瘤Daudi细胞对灭活流感病毒的敏感性。
Sensitivity of Burkitt lymphoma Daudi cells to inactive influenza virus.
作者信息
Shlomi Y, Zakay-Rones Z
机构信息
Department of Virology, Hebrew University-Hadassah Medical School, Jerusalem, Israel.
出版信息
J Cancer Res Clin Oncol. 1989;115(1):61-6. doi: 10.1007/BF00391601.
Abstract
Interaction of UV-inactivated influenza A/X47 virus at high multiplicity caused a rapid inhibition in cellular protein and DNA synthesis, thus arresting Burkitt-lymphoma-derived Daudi cell multiplication, and eventually killing the cells. The mechanism of the cytolytic effect is presumably, linked to the increase in cell membrane permeability indicated by elevation in 51Cr release. This might be the consequence of the mass adsorption and/or penetration of viral particles.
摘要
紫外线灭活的甲型/X47流感病毒在高感染复数下的相互作用导致细胞蛋白质和DNA合成迅速受到抑制，从而阻止了源自伯基特淋巴瘤的Daudi细胞增殖，并最终杀死细胞。细胞溶解作用的机制大概与51Cr释放增加所表明的细胞膜通透性增加有关。这可能是病毒颗粒大量吸附和/或穿透的结果。
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引用本文的文献
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Preferential cytotoxic effect of Newcastle disease virus on lymphoma cells.新城疫病毒对淋巴瘤细胞的优先细胞毒性作用。
J Cancer Res Clin Oncol. 1996;122(7):409-15. doi: 10.1007/BF01212880.
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J Cancer Res Clin Oncol. 1995;121(3):169-74. doi: 10.1007/BF01198099.
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J Cancer Res Clin Oncol. 1991;117(6):561-7. doi: 10.1007/BF01613289.
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